Titlebook: Biological Reactive Intermediates V; Basic Mechanistic Re Robert Snyder,I. Glenn Sipes,Charlotte M. Witmer Book 1996 The Editor(s) (if appl

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Muskelrelaxation bei alten Menschen,able as inducer of many immediate-early inflammatory and immune reactions, the transcription factor is likely to play a fatal role in certain diseases and syndromes that involve an abberrant expression of inflammatory cytokines [22–24].

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Muskelrelaxanzien und Leberinsuffizienz,ells. Interestingly, Ref-1 is a bifunctional protein; it also possesses an apurinic/apyrimidinic (A/P) endonuclease DNA repair activity. However, the redox and DNA repair activities of Ref-1 are physically and biochemically distinguishable. Ref-1 may represent a novel component of the signal transdu

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Muskelrelaxation bei alten Menschen,PK Ser/Thr kinase activity by both oxidant and antioxidant stimuli, which temporally correlates with the appearance of hyperphosphorylated TCF/Elk-1. Thus antagonistic intracellular redox changes can lead to the same functional modulation of the nuclear transcription factor TCF/Elk-1 that was previo

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0065-2598 d below, as well as the book for which this serves as the Preface. ? Jollow, DJ., Kocsis, J.J., Snyder, R. and Vainio, H. (eds), Biological Reactive Intermediat978-1-4757-9482-3978-1-4757-9480-9Series ISSN 0065-2598 Series E-ISSN 2214-8019

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Reactions of Reactive Metabolites with Hemoproteins—Toxicological Implicationsctive metabolites. It is known that at least three pathways exist for the covalent alteration of the cytochrome P450, one that involves alteration of the heme, a second that involves alteration of the protein, and a third that involves the crosslinking of heme to the protein (Scheme I) (Osawa & Pohl

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Identification of Hydrogen Peroxide as the Relevant Messenger in the Activation Pathway of Transcripable as inducer of many immediate-early inflammatory and immune reactions, the transcription factor is likely to play a fatal role in certain diseases and syndromes that involve an abberrant expression of inflammatory cytokines [22–24].