Titlebook: Eicosanoids and Other Bioactive Lipids in Cancer and Radiation Injury; Proceedings of the 1 Kenneth V. Honn,Lawrence J. Marnett,Thomas L. W

retort

書目名稱Eicosanoids and Other Bioactive Lipids in Cancer and Radiation Injury影響因子(影響力)




書目名稱Eicosanoids and Other Bioactive Lipids in Cancer and Radiation Injury影響因子(影響力)學科排名




書目名稱Eicosanoids and Other Bioactive Lipids in Cancer and Radiation Injury網(wǎng)絡(luò)公開度




書目名稱Eicosanoids and Other Bioactive Lipids in Cancer and Radiation Injury網(wǎng)絡(luò)公開度學科排名




書目名稱Eicosanoids and Other Bioactive Lipids in Cancer and Radiation Injury被引頻次




書目名稱Eicosanoids and Other Bioactive Lipids in Cancer and Radiation Injury被引頻次學科排名




書目名稱Eicosanoids and Other Bioactive Lipids in Cancer and Radiation Injury年度引用




書目名稱Eicosanoids and Other Bioactive Lipids in Cancer and Radiation Injury年度引用學科排名




書目名稱Eicosanoids and Other Bioactive Lipids in Cancer and Radiation Injury讀者反饋




書目名稱Eicosanoids and Other Bioactive Lipids in Cancer and Radiation Injury讀者反饋學科排名

胡言亂語

Transcellular Biosynthesis of Leukotrienes: Is Leukotriene A4 a Mediator? biochemical studies (1,2). In most cases, these studies involved the addition of radiolabeled arachidonic acid to homogenous cell suspensions with characterization of the resultant radiolabeled metabolites. Thus, most of the studies during the 1960s and 1970s were designed to establish those oxidat

prolate

Irrigate

Fatty Acid Cyclooxygenase Metabolism of Arachidonic Acid in Human Tumor Cells related to tumor metastasis, cell proliferation, tumor promotion and host immunoregulation (1–23). There is substantial evidence that human tumor cells may synthesize significant quantities of prostaglandins. Elevated production of prostaglandin E2 (PGE.) has been demonstrated in lung cancer patien

ICLE

影響帶來

Localization and Expression of the Eicosanoid Metabolizing P4501VA Gene Family in Rat Tissues and He cholesterol, arachidonic acid, leukotrienes and prostaglandins (2,3). Three genes of the rat 1VA gene family have been characterized and shown to be induced by tumor-promoting hypolipidemic drugs (2,3). The physiological function of the ω-oxidized products remains largely undefined. However, the ex

影響帶來

圍裙

licence

Prevention of Peroxide Produced DNA Strand Scission in Human Tumor Cells by Lipoxygenase Inhibitorseral orders of magnitude more toxic to cells at 37° C than at 0° C (1). At low doses, these agents produce mutation and transformation in various cells. The phorbol esters, transforming agents which are structurally unrelated to peroxide have also been reported to produce oxidative damage in cells.

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