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Titlebook: Cortical Deficits in Schizophrenia; From Genes to Functi Patricio O‘Donnell Book 2008 Springer-Verlag US 2008 DNA.Motivation.attention.beha

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31#
發(fā)表于 2025-3-27 00:24:09 | 只看該作者
Ulrich Schneekloth,Sabine Andresenfic psychophysiological deficits in sensory and motor gating, their neurobiological substrate in nicotinic acetylcholine-mediated neurotransmission, and their genetic basis. Detailed correlations between these several levels of observation are necessary to parse schizophrenia into discrete and hopef
32#
發(fā)表于 2025-3-27 01:49:30 | 只看該作者
Ulrich Schneekloth,Sabine Andresenansmission has a role in the ultimate clinical manifestations of this devastating illness. The glutamate hypothesis has evolved beyond the simplistic notion of NMDA receptor dysfunction, based in part on the dearth of consistently reproducible findings for altered NMDA receptor and binding site expr
33#
發(fā)表于 2025-3-27 07:35:09 | 只看該作者
34#
發(fā)表于 2025-3-27 12:13:25 | 只看該作者
35#
發(fā)表于 2025-3-27 14:44:51 | 只看該作者
36#
發(fā)表于 2025-3-27 20:27:00 | 只看該作者
37#
發(fā)表于 2025-3-28 00:48:14 | 只看該作者
Patricio O‘DonnellPresents the latest research on how genetic predisposition can produce schizophrenia’s pathophysiological outcomes
38#
發(fā)表于 2025-3-28 02:40:13 | 只看該作者
39#
發(fā)表于 2025-3-28 07:23:53 | 只看該作者
40#
發(fā)表于 2025-3-28 11:39:51 | 只看該作者
Ilene Klein CISSP, CISM, CIPP/USnt to which this heterogeneity reflects variability (variance of a particular parameter within a class) and diversity (the existence of different classes within a population) remains to be determined. As demonstrated by the other volumes in this book, multiple molecular, cellular and circuitry candidates could contribute to this heterogeneity.
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