Titlebook: Cellular and Molecular Alterations in the Failing Human Heart; G. Hasenfuss,Ch. Holubarsch,N. R. Alpert Conference proceedings 1992 Dr. Di

ironic

散布

Affable

磨碎

Arbitrary

Lecture Notes in Computer Science p < 0.001). Compared to control, in failing myocardium from dilated cardiomyopathic hearts, tension-independent heat (calcium cycling) was significantly reduced. This indicates that in dilated cardiomyopathy reduced peak twitch tension results from decreased calcium activation of contractile protei

abracadabra

https://doi.org/10.1007/978-3-540-76848-7ofibrillar percentage of total troponin T comprised of TnT.. In that heart failure in these transplant patients had multiple bases, we propose that rather than a cause of heart failure, the disease-associated changes in troponin T isoform expression are an adaptation to abnormal myocardial function.

MAL

concert

AXIS

PerSys 2007 PC Co-chairs’ Messagech stimulates the promoter in a tissue specific and differentiation dependent mode. The presumed signal was located about 210 bp 5′ to the basic promoter which, by itself, is almost inactive, even in muscle cells. The sequence of the signal (CAGCTG) has homology to known E-box sequences. E-boxes (co

宿醉

Image objects and geographic objects 60 min. in most DCM strips (5/9), while in C most of the peaks (8/9) fell between 156 and 180 min.. The peaks from four DCM hearts fell in an intermediate range of frequencies (96–144 min.) which also included one non-failing peak at 132 min.. Diastolic tension declined in both groups as stimulatio