Titlebook: Cellular and Molecular Alterations in the Failing Human Heart; G. Hasenfuss,Ch. Holubarsch,N. R. Alpert Conference proceedings 1992 Dr. Di

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Flu表流動

不近人情

朋黨派系

FLINT

Malfunction

Cardiovascular cyclic nucleotide phosphodiesterases and their role in regulating cardiovascular funcdence for the role that these isozymes have in the regulation of cellular processes has been generated through, or awaits, the identification of selective and potent PDE inhibitors. While selective inhibitors of the cGMP-inhibitable (cGi)-PDE isozyme have been approved for use in the acute treatment

Malfunction

Na,K-ATPase expression in normal and failing human left ventriclereparation for orthotopic transplantation or at the time of organ harvest. Abundance of mRNA for all three catalytic α subunits of the Na,K-ATPase was analyzed in samples from patients with end-stage heart failure due to either ischemic or dilated cardiomyopathy, as well as from normal controls. Van

CLAM

Structural and functional diversity of human ventricular myosinpressed, possible alterations in the myosin molecule could be of particular relevance. There is increasing evidence that myofibrillar ATPase activity is reduced in congestive heart failure, whereas the findings on myosin ATPase are still controversial. The molecular causes of the reduced activity ar

SPASM

Feckless