Titlebook: Anoikis; How the Extracellula Steven M. Frisch Book 2021 Springer Nature Switzerland AG 2021 Anoikis.Apoptosis.Cancer.Epithelial-Mesenchyma

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Metabolic Contributions to Anoikis-Resistance in Metastatic Dissemination,c and apoptogenic pathways converge on the mitochondria. Thus, accumulating evidence now links cellular metabolism after matrix detachment to both anoikis induction and anoikis resistance. Accordingly, the metabolic regulation of anoikis has become a topic of interest in cancer metastasis, since met

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The Intersection of Anoikis Resistance and Fatty Acid Metabolism in Cancer, cells enhance fatty acid oxidation activity to avoid anoikis and successfully colonize a secondary site. Targeting metabolic reprogramming and fatty acid oxidation is a feasible approach to inhibit cancer progression. Emphasizing ovarian cancer as a model system, this chapter will focus on the meta

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Anoikis and the Human Gut Epithelium in Health and Disease,indings concerning the regulation of anoikis in hIECs, as well as those pertaining to anoikis deregulation in CRC and IBD—with a partiality geared towards integrins, E-cadherins and the signaling they enact to this effect.

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Anoikis Mediated by Stress-Activated MAPK Signaling Pathways,ndantly to increase BIM and BMF apoptotic activity. The mechanism of cell death is mediated by the BAX/BAK-dependent intrinsic cell death pathway. Blocking either the JNK or the p38 MAPK pathway suppresses epithelial cell anoikis in vitro and prevents luminal clearance of mammary epithelial ducts in

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,The TGFβ and Androgen Receptor Signaling Pathways Converge to Support Anoikis Resistance in Triple tly studied in TNBC. Gene expression profiling combined with reverse phase protein array of TNBC cells in anchorage independent culture conditions revealed increased components of the TGFβ signaling pathway at the level of ligands, receptors and phosphorylated, nuclear Smad 3 localization and activi

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