Titlebook: Viruses and Apoptosis; Covadonga Alonso Book 2004 Springer-Verlag Berlin Heidelberg 2004 HIV.Herpes Virus.Viruses.apoptosis.cell.cell cycl

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Neuronal Apoptosis Pathways in Sindbis Virus Encephalitis,estruc-tion caused by their defensive campaign. Limited and localized responses are crucial to reduce the amount of “collateral damage” that may result in the elimination of vital components of the host essential for the survival of the infected organism. For instance, an excess in prophylactic prog

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Apoptotic Pathways Triggered By HIV and Consequences on T Cell Homeostasis and HIV-Specific Immunitthis syndrome was reported, it was discovered that the HIV envelope could bind to the CD4 receptor, that HIV could replicate within human CD4 T cells in vitro and kill them, and that circulating CD4 T cells decreased in number as disease progressed. Mature CD4 T helper cells are key effectors in coo

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HIV and Apoptosis: a Complex Interaction Between Cell Death and Virus Survival,as and Strasser 2000). The mechanisms of apoptosis are natural and even protect against uncontrolled cellular growth as well as playing a very important role in all development systems: embryogenesis, hematopoietic differentiation and proliferation, control of tumor proliferation, and regulation of

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Poliovirus and Apoptosis,ated vaccine were both developed in the 1950s (Salk 1955; Sabin and Boulger 1973), and subsequent massive vaccination campaigns resulted in near total eradication of PV from most industrialized countries: currently, wild strains are endemic in only 10 countries. However, there are two current proble

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Flaviviruses and Apoptosis Regulation,s ranging from mild febrile illnesses to severe hemorrhagic manifestations (yellow fever [YF], dengue [DEN]; Monath 2001; Guzman and Kouri 2002) or meningo-encephalitic syndromes (Japanese encephalitis [JE], Saint Louis encephalitis [SLE], West Nile [WN] fever and tickborne encephalitis [TBE]; Gould

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,Manipulation of Apoptosis by Herpes Viruses (Kaposi’s Sarcoma Pathogenesis), with clinical presentation of so-called pigmented sarcomas. This disease was designated Kaposi’s sarcoma (KS) in 1891. There are four distinct epidemiological forms of KS: classic KS occurring predominantly in Europe and the Mediterranean; endemic KS of the human immunodeficiency virus (HIV-1) nega

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Exploitation of Cell Cycle and Cell Death Controls by Adenoviruses: The Road to a Productive Infectability to deregulate these processes, they have contributed significantly to our knowledge of the underlying factors governing oncogenesis. In recent years, their recognized potential as therapeutic agents has led to a dramatic resurgence of interest in Adenoviruses. However, with this has come the

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Epstein-Barr Virus Signal Transduction and B-Lymphocyte Growth Transformation,cells into continuously proliferating lymphoblastoid cell lines (LCLs) while maintaining itself as an incomplete or “l(fā)atent” virus in the infected cell nucleus. Immune surveillance is critical to maintaining this symbiosis as perturbations to T-cell immunity reveal the malignant potential of latent

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