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Titlebook: Viral Heart Disease; Heinz-Dietrich Bolte Conference proceedings 1984 Springer-Verlag Berlin Heidelberg 1984 Antigen.Heart.Herzkrankheit.V

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發(fā)表于 2025-3-21 20:08:29 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Viral Heart Disease
編輯Heinz-Dietrich Bolte
視頻videohttp://file.papertrans.cn/984/983193/983193.mp4
圖書封面Titlebook: Viral Heart Disease;  Heinz-Dietrich Bolte Conference proceedings 1984 Springer-Verlag Berlin Heidelberg 1984 Antigen.Heart.Herzkrankheit.V
描述Since the last meeting "Myocardial Biopsy - Diagnostic Signifi- cance" was held under the auspices of the International Society and Federation of Cardiology (ISFC) in Munich, many new data and new aspects have been developed in the field of myocardial dis- eases. Most importantly, the classification of cardiomyopathies and specific heart muscle diseases, as described in the report of the WHO-ISFC task force (see reference 1 in the Introduction), is now widely accepted as a basis for clinical practice and scientific work. Investigators from all over the world have again assembled un- der the auspices of the ISFC at an international workshop on viral heart disease and its implications with regard to congestive cardio- myopathy, which was held in Munich January 1983. This book con- tains the significant results of the workshop. For practical use, the authors have incorporated important points made in the discussions into their contributions and a subject index has been provided. First of all I should like to thank Professor Riecker, Director of the Medizinische Klinik, Klinikum Grol3hadern, University of Mu- nich, West Germany. Without his friendly support in many aspects, this meetin
出版日期Conference proceedings 1984
關鍵詞Antigen; Heart; Herzkrankheit; Viruskrankheit; interferon
版次1
doihttps://doi.org/10.1007/978-3-642-95448-1
isbn_softcover978-3-540-13112-0
isbn_ebook978-3-642-95448-1
copyrightSpringer-Verlag Berlin Heidelberg 1984
The information of publication is updating

書目名稱Viral Heart Disease影響因子(影響力)




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書目名稱Viral Heart Disease網(wǎng)絡公開度學科排名




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Histomorphological Relations Between Myocarditis and Dilated Cardiomyopathyum is non-specifically thickened, particularly in long-standing cases. Thrombus may or may not be superimposed [1,2]. The coronary vessels are usually normal and the pericardium shows no abnormalities.
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The Role of Virus-, and Immune-Mediated Cardiocyte Injury in Coxsackievirus B3-Induced Myocarditisng RNA viruses with an icosahedral protein capsid. They are divided into the two major subgroups coxsackieviruses A and B with 24 and 6 serotypes respectively. While the group A viruses express distinct capsid antigens, the group B coxsackieviruses share a common group antigen [3, 18].
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The Role of Eosinophils in Myocarditishilis on heart muscle [2]. Other forms of heart disease in which eosinophils are prominent include parasitic [1], granulomatous [3] and hypersensitivity disorders [9], which usually present as an acute systemic illness.
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Viral Heart Disease — A Precursor of Congestive Cardiomyopathy been based on previous studies noting the persistence of symptoms and electrocardiographic and radiological abnormalities following acute viral myocarditis [2–5], as well as the demonstration of high viral antibody titers [6] and myocarditis by biopsy [7] in patients with the syndrome of dilated cardiomyopathy.
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