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Titlebook: Rheumatoid Arthritis; Recent Research Adva Josef S. Smolen,Joachim R. Kalden,Ravinder N. Main Conference proceedings 1992 Springer-Verlag B

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書目名稱Rheumatoid Arthritis
副標(biāo)題Recent Research Adva
編輯Josef S. Smolen,Joachim R. Kalden,Ravinder N. Main
視頻videohttp://file.papertrans.cn/831/830092/830092.mp4
圖書封面Titlebook: Rheumatoid Arthritis; Recent Research Adva Josef S. Smolen,Joachim R. Kalden,Ravinder N. Main Conference proceedings 1992 Springer-Verlag B
描述Many advances have occurred in the last 5 years especiallyin understanding genetic susceptibility factors, the role ofimmune response in chronicarthritis and the relationshipbetween cellular processes that underlie inflammation andtissue damage. All these are dealt with in depth intheindividual contributions.Genetics studies included illustrate howstudies of HLAgenes and genes controlling the autoantibody repertoiremaybe involved. Environmental factors are believed to beimportant and studies both in humans and in experimentalmodels on the role of viral andbacterial agents arecovered. The fascinating relationship betweenmicrobes,dysregulated immune responses and oncogenes are alsoexamined.Since autoimmunity is the key aspect of theimmunologicalresponse in rheumatoid arthritis, the role of antigensisre-examined as is the part played by immune T and B cells.New areas of clinical application of basic science is wellpresented in this book, forexample, in examining howmarkers of cartilage destruction may become important inmonitoring disease activity. A chapter reviewing possiblefuture novel therapies is included.
出版日期Conference proceedings 1992
關(guān)鍵詞Arthritis; Immungenetik; antibody; antigen; autoimmunity; bacteria; cartilage; genetics; immune response; imm
版次1
doihttps://doi.org/10.1007/978-3-642-76189-8
isbn_softcover978-3-642-76191-1
isbn_ebook978-3-642-76189-8
copyrightSpringer-Verlag Berlin Heidelberg 1992
The information of publication is updating

書目名稱Rheumatoid Arthritis影響因子(影響力)




書目名稱Rheumatoid Arthritis影響因子(影響力)學(xué)科排名




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Role and Regulation of Synovial MHC Class II Antigens in Rheumatoid Arthritis and Related Diseasesd synovial tissue indicate that local class II-dependent T cell activation can take place in synovial tissue [3–5]. Treatment with human immunoglobulin fractions enriched for anti-class II antibodies [6] as well as with mouse anti-CD 4 monoclonal antibodies [7] may diminish disease activity in RA.
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Conference proceedings 1992tis and the relationshipbetween cellular processes that underlie inflammation andtissue damage. All these are dealt with in depth intheindividual contributions.Genetics studies included illustrate howstudies of HLAgenes and genes controlling the autoantibody repertoiremaybe involved. Environmental f
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Streptococcal Cell Wall Antigens and Rheumatoid Arthritis 60, 62, 64]. Additional data link streptococcal cell wall proteins and related bacterial substances to rheumatoid factor production [9, 32, 42, 58]. Accordingly, this review will summarize some of the more recent information on these issues.
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