Titlebook: Recent Progress in Failing Heart Syndrome; Shigetake Sasayama (Professor of Medicine),Hiroyuk Book 1991 Springer-Verlag Tokyo 1991 adaptat

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Takeshi Nakatani,Hisateru Takano,Hiroyuki Noda,Masayuki Kinoshitaeptualizing infant-maternal attachment as an adaptation to usurpation. Distinctions are informed in terms of the adaptive advantages of exclusive (vs. nonexclusive) proximal contact with mother. Finally, we address milestones in socioemotional, social cognitive, and motor development that occur at 9

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achment, an affectional bond able to withstand being untethered to lactation by infants’ third year. The timing of the transition from lactation-based cohesion to attachment coincided with attenuated dependence on breast milk due to maturation of infants’ digestive and immune systems, and with the e

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Reconstitution of Actin-Myosin Interaction In Vitro: Its Application to Cardiac Physiology,scope was smooth and its velocity was related to the biochemical characteristics of myosin. The velocity changed under various experimental conditions, suggesting that the sliding observed in this system is physiological in nature..We consider that in vitro motility assay techniques can help us unde

出處

Molecular Basis for Cardiac Adaptation to Overload,d from these cDNA clones showed 91 and 96% homology, respectively. Using HCα and HCβ gene-specific sequences, we demonstrated that the transition of HCα to HCβ in the overloaded human heart was induced by the expression of HCβ gene..To determine the role of cellular oncogenes in the process of cardi

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Cardiac Oxygen Costs of Contractility (Emax) and Mechanical Energy (PVA): New Key Concepts in Cardiup or down in a parallel manner with enhancement or depression of E. by various inotropic interventions. These experimental findings can be described by the empirical equations Vo. = .PVA + ., and . = .E. + .. Coefficient . is the oxygen cost of PVA or mechanical energy, .PVA is the PVA-dependent Vo

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Myocardial Energetics of Hypertrophied Heart of Spontaneously Hypertensive Rat and Stroke-Prone Spond SHR-SP was impaired even when the coronary perfusion was sufficient. Our findings may contradict demonstrations of a shift in heavy chain myosin isozymes in hypertrophied small animal hearts, but may suggest that more drastic changes in the myocardium, which are disadvantageous to the myothermal