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Titlebook: Non-Neuronal Mechanisms of Brain Damage and Repair After Stroke; Jun Chen,John H. Zhang,Xiaoming Hu Book 2016 Springer International Publi

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發(fā)表于 2025-3-21 16:09:36 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱(chēng)Non-Neuronal Mechanisms of Brain Damage and Repair After Stroke
編輯Jun Chen,John H. Zhang,Xiaoming Hu
視頻videohttp://file.papertrans.cn/667/666982/666982.mp4
概述Discusses the roles of astrocytes, microglia, oligodendrocytes, endothelial cells, pericytes, and other immune cells.Explores which therapies hold promise for translation to humans.Only comprehensive
叢書(shū)名稱(chēng)Springer Series in Translational Stroke Research
圖書(shū)封面Titlebook: Non-Neuronal Mechanisms of Brain Damage and Repair After Stroke; Jun Chen,John H. Zhang,Xiaoming Hu Book 2016 Springer International Publi
描述This book provides a comprehensive overview of the latest research in the role of non-neuronal cells - astrocytes, oligodendrocytes, endothelial cells, pericytes, microglia, and other immune cells in ischemic brain injury and long-term recovery. In these cases, neurodegeneration and brain repair are controlled in a sophisticated system, incorporating interactions between different cell types and cellular systems. Also explored are the therapeutic strategies that target non-neuronal responses after stroke and their translational potentials.
出版日期Book 2016
關(guān)鍵詞blood brain barrier; brain repair; neuroinflammation; therapies; white matter
版次1
doihttps://doi.org/10.1007/978-3-319-32337-4
isbn_softcover978-3-319-81260-1
isbn_ebook978-3-319-32337-4Series ISSN 2363-958X Series E-ISSN 2363-9598
issn_series 2363-958X
copyrightSpringer International Publishing Switzerland 2016
The information of publication is updating

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https://doi.org/10.1007/978-3-319-32337-4blood brain barrier; brain repair; neuroinflammation; therapies; white matter
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Structural Alterations to the Endothelial Tight Junction Complex During Strokenctions (TJs) that link the cerebral endothelial cells are critical for BBB function and they undergo marked modifications after stroke. Those changes contribute to increased BBB permeability, brain edema, and leukocyte infiltration, all hallmarks of stroke. This review describes the types of TJ mod
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Role of Pericytes in Neurovascular Unit and Stroke that a successful neuroprotection is not feasible without microvascular protection. Recent studies on pericytes, long-neglected cells of the NVU have provided insight into the regulation of microcirculatory flow, blood–brain barrier (BBB) maintenance and angiogenesis. Ischemia induces persistent pe
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Glial Support of Blood–Brain Barrier Integrity: Molecular Targets for Novel Therapeutic Strategies istics (i.e., tight junction protein complexes, influx and efflux transporters) that control permeation of circulating solutes including drugs. In order to form this “barrier,” brain microvascular endothelial cells require support of adjacent astrocytes and microglia. This intricate relationship also
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Angiogenesis: A Realistic Therapy for Ischemic Strokeo various organs and tissue. Angiogenesis is strictly regulated by many key anti- or pro-angiogenic factors, and these molecules have been well-documented to play an important role in the development of angiogenesis in response to various pathological conditions. In human diseases angiogenesis has a
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