Titlebook: Melanoma; David E. Fisher,Boris C. Bastian Reference work 2019 Springer Science+Business Media, LLC, part of Springer Nature 2019 melanoma

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書目名稱Melanoma影響因子(影響力)




書目名稱Melanoma影響因子(影響力)學(xué)科排名




書目名稱Melanoma網(wǎng)絡(luò)公開度




書目名稱Melanoma網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Melanoma被引頻次




書目名稱Melanoma被引頻次學(xué)科排名




書目名稱Melanoma年度引用




書目名稱Melanoma年度引用學(xué)科排名




書目名稱Melanoma讀者反饋




書目名稱Melanoma讀者反饋學(xué)科排名

Analogy

Craig Sinclair,Louise F. Wilson,Catherine Olsen,Anna Nicholson

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Phagocytes

Developmental Biology of Melanocytesupon tumor initiation in a zebrafish model of melanoma, suggesting a functional involvement of a NCSC program in tumors originating from neural crest derivatives. Thus, to gain insights into melanoma biology, it is important to understand the mechanisms regulating NCSC and melanocyte development, as

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殺子女者

Key Signaling Pathways in Normal and Neoplastic Melanocyteskinase signaling are also frequent in melanoma and promote aberrant cell cycle progression. Other pathways such as Gαq, Wnt (canonical and noncanonical), Hippo, Notch, and signaling downstream of Rho family GTPases also play important roles in the aforementioned biological processes, and in some cas

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Melanoma Metabolismh suggests that the events required to transform cells to malignancy must allow cells to meet their biosynthetic and bioenergetic needs. During progression, melanomas appear to display metabolic heterogeneity as vital nutrient sources become scarce and the tumor seeks to overcome these restrictions.

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Principles of Targeted Therapyment. ERK inhibitors are currently undergoing clinical development. KIT mutations are primarily found in acral and mucosal melanomas, and several KIT inhibitors have been tested in KIT-mutant melanoma clinical trials; so far none have been approved by regulatory agencies. The challenge for KIT inhib