Titlebook: Mechanisms of Anesthetic Action in Skeletal, Cardiac, and Smooth Muscle; Thomas J. J. Blanck,David M. Wheeler Book 1991 The Editor(s) (if

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Ca Release from Skeletal Muscle SRed, probably due to different experimental models and different concentrations of the drugs used. Often these studies address volatile anesthetic effects at concentrations well above those required for clinical anesthesia.

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Interactions of Fatty Acids with the Calcium Release Channel in Malignant Hyperthermiabased on muscle rigidity during the syndrome and the increase in Ca. in isolated fiber bundles exposed to halothane. Additionally, the threshold of Ca.-induced Ca. release (TCICR) is lower than normal in isolated fractions of heavy sarcoplasmic reticulum (HSR) from porcine MH muscle.. However, the d

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Cardiac Effects of Anestheticsigned to examine the effects of anesthetics on ionic fluxes across the sarcolemma, rapid changes in intracellular calcium, calcium transport functions of the sarcoplasmic reticulum (SR), isometric contractile force, and calcium sensitivity of the contractile apparatus.

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Effects of Volatile Anesthetics on the Intracellular Calcium Transient and Calcium Current in Cardiaant decrement of cardiovascular homeostasis. The mechanism of action underlying the negative inotropic effect of volatile anesthetics is not fully understood. There are several mechanisms by which agents may directly alter contractile performance of cardiac muscle. The first group represents the “up

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Halothane Inhibits Binding of Calcium Channel Blockers to Cardiac Sarcolemmaeostasis of the myocardial cell.. Our laboratory has examined three important sites in the myocardial cell that are known to be involved with the phasic changes in intracellular calcium which occur during contraction. These sites are the sarcolemma, the sarcoplasmic reticulum, and the myofibrils. Th

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Contribution of the Known Subcellular Effects of Anesthetics to Their Negative Inotropic Effect in Ilative importance of these multiple effects to the overall negative inotropic response in intact myocardium. We have used isolated rabbit papillary muscles and left atrial muscles to evaluate the relative contribution of anesthetic effects on the transsarcolemmal Ca. influx, on the function of the s

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