Titlebook: Lupus; Molecular and Cellul Gary M. Kammer,George C. Tsokos Book 1999 Springer Science+Business Media New York 1999 Antigen.apoptosis.autoi

formation

書目名稱Lupus影響因子(影響力)




書目名稱Lupus影響因子(影響力)學(xué)科排名




書目名稱Lupus網(wǎng)絡(luò)公開度




書目名稱Lupus網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Lupus被引頻次




書目名稱Lupus被引頻次學(xué)科排名




書目名稱Lupus年度引用




書目名稱Lupus年度引用學(xué)科排名




書目名稱Lupus讀者反饋




書目名稱Lupus讀者反饋學(xué)科排名

小步走路

AUGUR

忙碌

Genetics of Systemic Lupus Erythematosus,the promise of enhanced insights to be gained from the study of spontaneous and induced autoimmune disease in animals. However, this enthusiasm is tempered by the fact that nearly two decades after particular major histocompatibility complex (MHC) alleles were found to be associated with SLE, we sti

不可思議

Lessons from Knockout and Transgenic Lupus-Prone Mice,umbers of T cells unable to undergo Fas-induced cell death after activation. Nevertheless, such animals with known genetic alterations provide substantially increased insight into the lupus immune system by allowing an examination of disease manifestations in the setting of known immune deficiencies

歸功于

B Cells in Systemic Lupus Erythematosus,ven though questions always arise whether the derived conclusions can be transferred unmodified to human disease. This chapter focuses on the contribution of B cells to the pathogenesis of human SLE, and makes reference to the murine models of lupus to clarify our understanding of the autoimmune B c

nascent

T Cell Autoimmunity in Lupus,identical to those important in experimental models of lupus autoimmunity. For example, autoimmune-prone mice depleted of either B or T lymphocytes fail to exhibit the typical immunologic or pathologic outcomes of this disease .. However, these mice also fail to respond to pathogens or conventional

Armory

外貌

STERN

Natural Killer Cells and CD8+ T Cells in the Downregulation of Antibody Production in Healthy Subjeroduction have been described. The identity and characterization of antigen nonspecific regulatory cells has been a principal interest of our group. Whereas Stohl has focused his attention on the role of cytolytic T cells in SLE (. Chapter 18), we have contemplated how antigen nonspecific suppressor