Titlebook: Liver Growth and Repair; Alastair Strain,Anne Mae Diehl Book 1998 Springer Science+Business Media Dordrecht 1998 cells.cyclin.development.

deep-sleep

書目名稱Liver Growth and Repair影響因子(影響力)




書目名稱Liver Growth and Repair影響因子(影響力)學(xué)科排名




書目名稱Liver Growth and Repair網(wǎng)絡(luò)公開度




書目名稱Liver Growth and Repair網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Liver Growth and Repair被引頻次




書目名稱Liver Growth and Repair被引頻次學(xué)科排名




書目名稱Liver Growth and Repair年度引用




書目名稱Liver Growth and Repair年度引用學(xué)科排名




書目名稱Liver Growth and Repair讀者反饋




書目名稱Liver Growth and Repair讀者反饋學(xué)科排名

懶洋洋

978-94-010-6069-1Springer Science+Business Media Dordrecht 1998

AMOR

Proto-oncogenes/ transcription factors of these events are presumably intimately linked with the eventual regeneration of the liver, whereas others are likely to be stress-related or required for the continued differentiated function of the liver while regeneration is occurring.

ARY

Proteoglycansll-matrix and cell-cell adhesion, control of extracellular enzymic activities and the regulation of cell growth itself, through modulation of growth factor activation (for reviews see Gallagher, 1989; Couchman and Woods, 1993).

tic-douloureux

in vivoRecent molecular biology has enabled remarkable progress in biological research. However, this biological achievement requires evaluation by different approaches, particularly in the understanding of complexity in dynamic organs. Such a situation exists exactly in the regulatory mechanisms of liver regeneration.

HIKE

CytokinesLiver growth occurring after liver damage or partial hepatectomy is a well-known phenomenon in the wide field of hepatology. While liver repair may involve repair mechanisms also observed in other organs, it is characteristic of the liver to generate the previous organ mass after partial hepatectomy in a very short time.

神化怪物

植物茂盛

Ordeal

Liver regeneration following hepatic injuryd hepatocellular proliferation in the . model of partial hepatectomy. In contrast, the characteristics and mechanisms of hepatic regeneration stimulated by liver cell loss from hepatocyte injury and cell death rather than surgical resection are largely unknown. The reasons for this imbalance in our

peptic-ulcer

Epithelial stem-like cells of the rodent liverence that has accumulated from nearly 100 years of investigation into the biology of the liver. Early in this century, investigators recognized potential lineage relationships among biliary epithelial cells, transitional cells and hepatocytes in several rodent models of liver regeneration (MacCallum