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Titlebook: Konjunkturschwankungen; Theorie, Messung, Pr Gunther J. Tichy Textbook 19761st edition Springer-Verlag Berlin Heidelberg 1976 Besch?ftigung

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發(fā)表于 2025-3-21 19:17:43 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Konjunkturschwankungen
副標(biāo)題Theorie, Messung, Pr
編輯Gunther J. Tichy
視頻videohttp://file.papertrans.cn/546/545280/545280.mp4
叢書名稱Heidelberger Taschenbücher
圖書封面Titlebook: Konjunkturschwankungen; Theorie, Messung, Pr Gunther J. Tichy Textbook 19761st edition Springer-Verlag Berlin Heidelberg 1976 Besch?ftigung
出版日期Textbook 19761st edition
關(guān)鍵詞Besch?ftigung; Depression; Einkommen; Einkommensverteilung; Keynes; Konjunktur; Konjunkturprognose; Konjunk
版次1
doihttps://doi.org/10.1007/978-3-642-96306-3
isbn_ebook978-3-642-96306-3Series ISSN 0073-1684
issn_series 0073-1684
copyrightSpringer-Verlag Berlin Heidelberg 1976
The information of publication is updating

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發(fā)表于 2025-3-21 21:21:56 | 只看該作者
Gunther J. Tichy the insulin-like factors with other growth factors on these responses has continued to be an interest of our laboratory. In addition, studies from our group (6), as well as more numerous data from others represented at this symposium, indicated that the IGFs had stimulatory effects on the cytodiffe
板凳
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地板
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發(fā)表于 2025-3-22 09:58:51 | 只看該作者
Gunther J. Tichy of tumor formation are often inhibited in proliferation by TGFβ later stages of tumor progression are positively regulated by TGFβ Accordingly, elements of TGFβ signal transduction are mutated in some tumor types while TGFβ signaling is maintained and activated in the majority of other tumors. TGFβ
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發(fā)表于 2025-3-22 13:08:09 | 只看該作者
Gunther J. Tichynally prepared as a competitive antagonist for HGF, is an internal fragment of HGF and it contains N-terminal hairpin and four kringle domains. NK4 binds to but does not activate the c-Met receptor, thereby competitively antagonizing biological activities of HGF. Unexpectedly, NK4 was subsequently s
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Gunther J. Tichydiated by estrogen and progesterone (again under pituitary control) (2). In rodent models of carcinogen-induced breast cancer, it has been shown that both progesterone and estrogen seem to be required for tumor formation and for early growth of tumors (3–4). Presumably, the mechanism of interaction
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發(fā)表于 2025-3-23 03:18:57 | 只看該作者
Gunther J. Tichy mullerian duct regression (3–4). Testosterone can facilitate this activity of MIS, but cannot, on its own, direct this important event in the development of the male phenotype. Although the fetal ovary does not produce MIS, it has recently been shown that the granulosa cells of the young and adult
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發(fā)表于 2025-3-23 06:35:16 | 只看該作者
Gunther J. Tichyrnell. Subsequently, we published a full description of our findings (7–8). It was at this point that we asked the question: What ovarian factor might retard the growth of endothelial cells in vitro? In searching the literature we found a publication by Sato, et al., 1986, that described morphologic
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