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Titlebook: Intracranial Pressure III; Proceedings of the T Johannes W. F. Beks,D. Andries Bosch,Mario Brock Conference proceedings 1976 Springer-Verla

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書目名稱Intracranial Pressure III
副標(biāo)題Proceedings of the T
編輯Johannes W. F. Beks,D. Andries Bosch,Mario Brock
視頻videohttp://file.papertrans.cn/474/473149/473149.mp4
圖書封面Titlebook: Intracranial Pressure III; Proceedings of the T Johannes W. F. Beks,D. Andries Bosch,Mario Brock Conference proceedings 1976 Springer-Verla
描述The papers presented at the Third International Symposium on Intracranial Pressure that was held at the University of Groningen. June 1-3, 1976 are brought together in this volume. After the successful meetings in Hannover and Lund the feeling of the Advisory Board was. that the third meeting should have more clinical implications. and therefore 7 out of the 10 Sessions deal with clinical problems. That there is still a growing interest in the problems of the intracranial pressure appears from the fact that 152 papers were submitted. Unfortunately only 56 could be read. due to lack of time. The selection was made by the members of the Advisory Board. It is a pity that so many good papers had to be rejected because they did not fit in the program. It was decided in a meeting of chairmen and co-chairmen during the symposium that the next congress will take place in 1979. to keep away from the CBF-meeting in June 1977 in Copenhagen and the "Brain-Edema" meeting in 1979 in Bethesda. This fourth symposium will be held in Williamsburg (USA) and prepared by Donald P. Becker. Douglas Miller. Gerald M. Hochwald and Kenneth Shulman.
出版日期Conference proceedings 1976
關(guān)鍵詞Hirndruck; Hydrocephalus; aneurysm; brain; brain injury; care; feeling; head injury; intracranial pressure; m
版次1
doihttps://doi.org/10.1007/978-3-642-66508-0
isbn_softcover978-3-642-66510-3
isbn_ebook978-3-642-66508-0
copyrightSpringer-Verlag Berlin · Heidelberg 1976
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The Influence of Brain Tissue Pressure Upon Local Cerebral Blood Flow in Vasogenic Edemacal cerebral blood flow (CBF) is unknown. The local CBF represents the generating source of the edema seen in this model. Our objective in the present study was to determine the temporal response of local white matter CBF to vasogenic edema and correlate these changes with parallel studies of local
地板
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Pathogenetic Role of the No-Reflow Phenomenon in an Experimental Subarachnoid Hemorrhage Model in thage of SAH (4). Since the result of SAH has been shown to depend on the mechanism of genesis of this anoxic-ischemic lesion of the brain is of great clinical importance. In the past, cerebral vasospasm has been regarded as a major factor in this respect (1, 8). However, most of the recent clinical a
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Intracranial Blood Volume and Its Variation with Changes in Intracranial Pressure changes in the ICBV on intracranial pressure (ICP) are magnified by intracranial mass lesions and increased pressure. Under extreme conditions with cerebral vasoparalysis the vascular bed may assume excessive dimensions with adverse consequences (2). An accurate assessment of the mechanical effects
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Differences in the Behavior of Afferent Vessels in Autoregulation to Increased Intracranial Pressure capacity. Both decrease the transmural pressure across the cerebral resistance vessels, and it has seemed reasonable to assume a probable identity of mechanisms for autoregulation in the two circumstances, at any rate as far as the intracranial vessels have been concerned.
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Changes of the Sympathetic Vasomotor Activity During Increased Intracranial Pressurewaves during increases in intracranial pressure (ICP), the precise mechanisms remain unclear. Therefore, in the present study the direct observation of the sympathetic vasomotor activity during increases in ICP was attempted by measuring the efferent discharge from the renal nerve of the cat.
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