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Titlebook: Glutamate and Neuropsychiatric Disorders; Current and Emerging Zoran M. Pavlovic Book 2022 Springer Nature Switzerland AG 2022 Aggression.A

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發(fā)表于 2025-3-23 10:29:25 | 只看該作者
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發(fā)表于 2025-3-23 19:10:39 | 只看該作者
https://doi.org/10.1007/978-3-662-55990-1ion. As such, there is great interest in identifying optimal treatment options which beneficially reduce pain with limited side effects. Potential options include antagonism of ionotropic and Group I metabotropic glutamate (mGlu) receptors, agonism of Group II and III mGlu receptors, modulation of t
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發(fā)表于 2025-3-24 01:40:06 | 只看該作者
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發(fā)表于 2025-3-24 02:31:26 | 只看該作者
Einleitung: Fragestellung und Studiendesign,ous system (CNS). Besides these signs, a diffuse loss of synaptic contacts, axonal pruning and astrocytosis are also observed, that in general correlate with the dysregulation of the glutamatergic system and with the onset of neurological symptoms. Concomitantly to the synaptic derangements, impaire
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發(fā)表于 2025-3-24 07:16:56 | 只看該作者
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發(fā)表于 2025-3-24 11:28:56 | 只看該作者
https://doi.org/10.1007/978-3-658-17617-4logy of schizophrenia. Genetic, postmortem and neuroimaging studies in schizophrenia patients showed alterations in different glutamatergic elements. Thus, glutamatergic system has emerged as a promising target for the treatment of schizophrenia, especially for symptoms not addressed by current anti
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發(fā)表于 2025-3-24 18:54:39 | 只看該作者
Leben mit der koronaren Herzkrankheitalance can induce neuronal hyper-excitability in a susceptible neuronal network leading to seizures. Glutamate is the main excitatory neurotransmitter in the nervous system but when it is accumulated in excess induces neurotoxicity, mediates neuronal hyper-excitability and seizures. In epilepsy, glu
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發(fā)表于 2025-3-24 21:43:56 | 只看該作者
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