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Titlebook: Genomic Instability and Immortality in Cancer; Enrico Mihich,Leland Hartwell Book 1997 Springer Science+Business Media New York 1997 DNA.M

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發(fā)表于 2025-3-23 11:02:31 | 只看該作者
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發(fā)表于 2025-3-23 14:34:22 | 只看該作者
Praktische Hilfen zur Lebensgestaltung,ticipated both the importance of genetic instability in the initiation and progression of cancer and the existence of negative regulators of cell growth, whose loss would result from inappropriate chromosome segregation.
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發(fā)表于 2025-3-23 18:06:22 | 只看該作者
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發(fā)表于 2025-3-24 00:26:58 | 只看該作者
Zusammenfassung, Diskussion und Ausblick,ls arrest late in G1 when p53 is active. In addition to G1 arrest, regulation by p53 of the other major cell cycle checkpoint, G2/M, has recently been reported in two different systems (Stewart ., 1995; Agarwal .., 1995).
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發(fā)表于 2025-3-24 04:20:08 | 只看該作者
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發(fā)表于 2025-3-24 06:42:51 | 只看該作者
Hinweise zum Gebrauch des Glossars, 8–12 bases of unreplicated DNA at the terminus. Cells avoid this loss by utilizing a specialized telomeric DNA replication enzyme called telomerase that specifically recognizes telomeric DNA as a substrate. Telomerase is a unique reverse transcriptase that utilizes an RNA template to create a singl
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https://doi.org/10.1057/978-1-137-57889-1d carcinomas in surgical specimens. Although the study of advanced cancers is useful for identifying genetic abnormalities that have accumulated in the cancer, the ability of this approach to determine the order in which genetic and other abnormalities develop during earlier stages of neoplastic pro
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發(fā)表于 2025-3-25 00:26:23 | 只看該作者
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