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Titlebook: Genetics of Influenza Viruses; Peter Palese,David W. Kingsbury Book 1983 Springer-Verlag/Wien 1983 Grippeviren.Molekulargenetik.Viruses.ge

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發(fā)表于 2025-3-21 19:48:32 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Genetics of Influenza Viruses
編輯Peter Palese,David W. Kingsbury
視頻videohttp://file.papertrans.cn/383/382735/382735.mp4
圖書封面Titlebook: Genetics of Influenza Viruses;  Peter Palese,David W. Kingsbury Book 1983 Springer-Verlag/Wien 1983 Grippeviren.Molekulargenetik.Viruses.ge
描述With the advent of genetic engineering methods and improved biochemical tech- niques, much has been learned about the replication of influenza viruses, their structure and their epidemiology. It appears that the time is ripe to review these efforts and to provide a molecular perspective of influenza virology. It is hoped that this book will stimulate our thinking, help us in designing new experiments, and possibly show avenues leading to the control of the diseases associated with influenza viruses. Peter Palese, New York, N. Y. August 1983 David W. Kingsbury, Memphis, Tenn. Contents List of Contributors. . . . . . . . . . . . . . . . . XV 1. The Evolution of Influenza Viral Genetics - A Perspective. By E. D. Kilbourne. . . . . . . . . . . . . . . . 1 I. Introduction. . . . . . . . . . . . . . . . . 1 II. The Development of Modern Influenza Viral Genetics 2 A. Early Evidence of Genetic Variation in the Laboratory 2 B. Application of Formal Genetic Techniques to Studies of Influenza Virus . . . . . . . 3 C. Genetic Markers. . . . . . . . . 3 D. Development of Plaquing Systems. . . 4 E. The Use of Conditional Lethal Mutants 5 F. New Approaches in Influenza Virus Genetics. 6 1. The Bi
出版日期Book 1983
關(guān)鍵詞Grippeviren; Molekulargenetik; Viruses; genetics; molecular genetics
版次1
doihttps://doi.org/10.1007/978-3-7091-8706-7
isbn_softcover978-3-7091-8708-1
isbn_ebook978-3-7091-8706-7
copyrightSpringer-Verlag/Wien 1983
The information of publication is updating

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https://doi.org/10.1007/978-3-540-85251-3 the clinical and epidemiological features of past and modern epidemics are so similar, we can assume that past epidemics were caused by antigenically variable viruses like contemporary strains now under study. The legitimacy of this assumption is strengthened by serologic, and more recently, molecu
地板
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https://doi.org/10.1007/978-3-531-19508-7le-stranded RNA genome which has been called “negative stranded” because the viral messenger RNAs are transcribed from the viral RNA segments. A great deal of new knowledge has been obtained about influenza A, B, and C viruses since the last major reviews of influenza virus in 1975 (Kilbourne, 1975)
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Die Methoden des Anbaus im allgemeinen,NA) and the virion contains the enzyme system which transcribes the NA into the viral NA [75]. The synthesis of influenza viral NA involves a unique interaction with the host cell transcriptional machinery in the nucleus of the infected cell. This interaction is required first for the initiation of
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https://doi.org/10.1007/978-3-476-03238-6tain only 7 segments (for reviews see Palese, 1977; Scholtissek, 1978, 1979a; Palese ., 1980). This unusual structure of the influenza virus genome makes a genetic comparison of different isolates interesting, because during double infection of a host with two different strains of the same type, eac
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Das Herumkutschieren auf der Modus-Karte,nted expansion of our knowledge about the proteins of the virus. Analysis of the DNA sequence of the cloned genes, added to the information available from earlier classical protein chemistry studies, has led to the elucidation of the amino acid sequences of all the known virus-coded proteins and to
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