Titlebook: Genetics of Influenza Viruses; Peter Palese,David W. Kingsbury Book 1983 Springer-Verlag/Wien 1983 Grippeviren.Molekulargenetik.Viruses.ge

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https://doi.org/10.1007/978-3-540-85251-3 the clinical and epidemiological features of past and modern epidemics are so similar, we can assume that past epidemics were caused by antigenically variable viruses like contemporary strains now under study. The legitimacy of this assumption is strengthened by serologic, and more recently, molecu

江湖郎中

https://doi.org/10.1007/978-3-531-19508-7le-stranded RNA genome which has been called “negative stranded” because the viral messenger RNAs are transcribed from the viral RNA segments. A great deal of new knowledge has been obtained about influenza A, B, and C viruses since the last major reviews of influenza virus in 1975 (Kilbourne, 1975)

Bridle

Die Methoden des Anbaus im allgemeinen,NA) and the virion contains the enzyme system which transcribes the NA into the viral NA [75]. The synthesis of influenza viral NA involves a unique interaction with the host cell transcriptional machinery in the nucleus of the infected cell. This interaction is required first for the initiation of

gastritis

https://doi.org/10.1007/978-3-476-03238-6tain only 7 segments (for reviews see Palese, 1977; Scholtissek, 1978, 1979a; Palese ., 1980). This unusual structure of the influenza virus genome makes a genetic comparison of different isolates interesting, because during double infection of a host with two different strains of the same type, eac

gastritis

CUMB

Das Herumkutschieren auf der Modus-Karte,nted expansion of our knowledge about the proteins of the virus. Analysis of the DNA sequence of the cloned genes, added to the information available from earlier classical protein chemistry studies, has led to the elucidation of the amino acid sequences of all the known virus-coded proteins and to

jagged

Desert