Titlebook: Genetic Instability and Tumorigenesis; Michael B. Kastan Book 1997 The Editor(s) (if applicable) and The Author(s), under exclusive licens

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書目名稱Genetic Instability and Tumorigenesis影響因子(影響力)




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書目名稱Genetic Instability and Tumorigenesis網(wǎng)絡(luò)公開度學(xué)科排名




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書目名稱Genetic Instability and Tumorigenesis被引頻次學(xué)科排名




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0070-217X c cells which con- tribute to the transformed phenotype arise from DNA damage caused by such exposures. Therefore, understanding how cells respond to DNA-damagi978-3-642-64434-4978-3-642-60505-5Series ISSN 0070-217X Series E-ISSN 2196-9965

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Pathogenese des postthrombotischen Syndroms,w will focus on recent advances in cell cycle control and genetic instability in the budding yeast ., with emphasis on experimental topics in which loss of cell cycle progression control decreases the fidelity of chromosome transmission to daughter cells. Viable single gene mutations that perturb bo

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collagenase

Genomic Instability and Its Role in Neoplasia,ade surrounding tissues, enter the vasculature, extravasate, and colonize a secondary site. Proficiency at each step is necessary for a tumor cell to become fully metastatic. Genetic alterations are the basis for this acquired variation (. 1987; . et al. 1987). The emergence of drug-resistant or rad

collagenase

https://doi.org/10.1007/978-3-531-90750-5 diseases, we think about inheriting one or two abnormal genes from our parents and these gene abnormalities confer the disease phenotype. In contrast, in the majority of cancers, no such inherited gene abnormalities can be identified (which does not mean that they do not exist) and there is no obvi

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Utho Creusen,Gordon Müller-Seitzon. New concepts make us realize the magnitude of the challenge of duplicating and distributing an accurate copy of the genome each time a cell divides. For each of the 10. somatic cell divisions that occur in a human lifetime, each copy of the genome must contain an intact sequence of some 3 billio

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depreciate