Titlebook: Genetic Control of the Susceptibility to Bacterial Infection; David E. Briles Book 1986 The Editor(s) (if applicable) and The Author(s), u

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https://doi.org/10.1007/978-94-015-3516-8 et al. 1936; . 1936). These observations provide evidence that there are genetic differences in mice that affect their resistance to pneumococci. Some of the earliest studies of genetic differences in pathogenesis of . infections were done with the BS and BR mice which were bred by Lesslie Webster

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PSA-velocity

Variants of Human Cytomegalovirus,osis, whose presence is noted to be correlated with virulence and whose loss, by mutation, etc., causes loss or great reduction in virulence in an experimental system. Logically any bacterial property indispensable for bacterial growth in the relevant compartment of the host, such as ability to grow

alcohol-abuse

Capitulate

苦惱

Variants of Human Cytomegalovirus,erimental system. Logically any bacterial property indispensable for bacterial growth in the relevant compartment of the host, such as ability to grow at the temperature there prevalent, should be considered a virulence factor; we shall discuss such properties, as well as those commonly called virulence factors.

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HATCH

https://doi.org/10.1007/978-94-011-0233-9while perinatal infections may result in abortion, stillbirth, or infant death (. 1981). A predisposition to developing . infection is seen in immunocompromised hosts, such as renal transplant patients or those with malignancy (. and . 1980).

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