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Titlebook: Genetic Control of the Susceptibility to Bacterial Infection; David E. Briles Book 1986 The Editor(s) (if applicable) and The Author(s), u

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書目名稱Genetic Control of the Susceptibility to Bacterial Infection
編輯David E. Briles
視頻videohttp://file.papertrans.cn/383/382466/382466.mp4
叢書名稱Current Topics in Microbiology and Immunology
圖書封面Titlebook: Genetic Control of the Susceptibility to Bacterial Infection;  David E. Briles Book 1986 The Editor(s) (if applicable) and The Author(s), u
出版日期Book 1986
關(guān)鍵詞bacteria; diseases; gene; gene regulation; genetics; immune system; immunodeficiency; infection; infections;
版次1
doihttps://doi.org/10.1007/978-3-642-70986-9
isbn_softcover978-3-540-16238-4
isbn_ebook978-3-642-70986-9Series ISSN 0070-217X Series E-ISSN 2196-9965
issn_series 0070-217X
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH, DE
The information of publication is updating

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Michael S. Rolph,Stefan H. E. Kaufmannibility complex (H-2) on the susceptibility or resistance of different strains of mice to a variety of diseases including bacterial infections. As a result, numerous investigations have resulted in a veritable explosion of knowledge concerning these interrelationships. Many aspects of these murine H
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https://doi.org/10.1007/978-1-4613-1137-9do this is to identify genes of the host that affect the resistance to the pathogen and then determine their mechanisms of action. This approach automatically establishes the in vivo relevance of all mechanisms studied. the discovery of variant alleles affecting particular functions also serves to i
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Cytokines in Hemopoiesis, Oncology, and AIDSate is ≥10. bacteria (. 1960; . and . 1974a; . 1982) unless the animals are pretreated with iron and/or an iron chelater (. et al. 1980; . 1982) or the mice are challenged intraperitoneally with . suspended in hog gastric mucin (. et al. 1936; . 1964). By contrast, . primarily evokes gastroenteritis
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Gynecological Applications of Cytology,other-sister mating, the mice derived from this litter, which had normal vigor and reproductive capacity under laboratory conditions, were considered to be inbred. In the early 1970s, it was shown that these mice made abnormal immune responses to polysaccharide antigens (. et al. 1972; . et al. 1973
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