Titlebook: Etiology of Hodgkin’s Disease; Ruth F. Jarrett Book 1995 Springer Science+Business Media New York 1995 Antigen.Pathogene.epidemiological.e

Plaque

,The Nature of the Lymphocytes in Hodgkin’s Disease,

現(xiàn)實(shí)

思想

https://doi.org/10.1007/978-1-4302-1941-5sis by determining the frequency of HLA-DPB1 alleles in two series of HD patients using molecular typing methods. One series consisted of a retrospective/prospective group of 118 patients over the age of 15 years, and the other a prospective group of 45 patients between the ages of 16 and 24 years.

Encumber

https://doi.org/10.1007/978-1-4302-1832-6haryngeal carcinoma, where the interaction of genetic and environmental factors is important in producing the observed pattern of disease. In the case of malignant melanoma, for example, a subset of the population has been identified with hereditary predisposition to melanoma associated with dysplas

證實(shí)

https://doi.org/10.1007/978-3-319-54879-1 EBV infection usually occurs in childhood and, once infected, individuals become life-long virus carriers. The virus is the causative agent of infectious mononucleosis (IM), a self-limiting lymphoproliferative disease resulting from delayed primary EBV infection, and is also associated with a numbe

Cholagogue

https://doi.org/10.1057/9780230374188 8 years that a firm molecular basis for this association has been established. The literature relating to EBV and HD has increased exponentially over the latter years and laboratories throughout the world have contributed to a body of data consistently linking this virus with HD. The majority of pa

似少年

Development Administration in the Caribbeanive” cells. The origin of the neoplastic cells in HD is still controversial (Hsu .., 1985; Cabanillas .., 1988; Hsu ..., 1992; Drexler, 1992). Recent data favour the hypothesis that the RS cells and their mononuclear variants (collectively called H-RS cells) originate from lymphoid cells, although c

Perennial長(zhǎng)期的

https://doi.org/10.1057/9780230595163genomes or gene products cannot be detected within the affected tissues of all cases. We have designated those cases in which clonal EBV genomes are detectable within tumours or in which the Reed-Sternberg (RS) cells express EBV LMP-1 protein or EBER RNAs as EBV-associated (Armstrong .., 1992). Stud

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