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Titlebook: Etiology of Hodgkin’s Disease; Ruth F. Jarrett Book 1995 Springer Science+Business Media New York 1995 Antigen.Pathogene.epidemiological.e

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書(shū)目名稱Etiology of Hodgkin’s Disease
編輯Ruth F. Jarrett
視頻videohttp://file.papertrans.cn/317/316393/316393.mp4
叢書(shū)名稱NATO Science Series A:
圖書(shū)封面Titlebook: Etiology of Hodgkin’s Disease;  Ruth F. Jarrett Book 1995 Springer Science+Business Media New York 1995 Antigen.Pathogene.epidemiological.e
描述This volume reports the proceedings of a NATO Advanced Workshop held at Cameron House Hotel, Loch Lomond, Scotland, from May 2 - 5, 1994. The major impetus for this workshop was the realisation, over the past 7 years, that the Epstein-Barr virus is associated with a proportion of cases of Hodgkin‘s disease and is likely to play an aetiological role. There were four main aims of the workshop: first, to discuss the recent findings in relation to Epstein- Barr virus and the aetiology of Hodgkin‘s disease; second, to relate these data to the epidemiology of Hodgkin‘s disease; third, to discuss other potential aetiological factors and finally, to discuss future directions for research into Hodgkin‘s disease. Leading experts in the field have contributed chapters to this volume. There is some overlap among chapters, particularly regarding Epstein-Barr virus, thereby allowing different groups to express views on similar topics. Perhaps, however, the most surprising feature of the workshop was the lack of controversy regarding the role of Epstein-Barr virus in Hodgkin‘s disease, an association that was treated with great scepticism at the beginning of the decade. The first three chapters,
出版日期Book 1995
關(guān)鍵詞Antigen; Pathogene; epidemiological; epidemiology; transplantation
版次1
doihttps://doi.org/10.1007/978-1-4613-0339-8
isbn_softcover978-1-4613-8005-4
isbn_ebook978-1-4613-0339-8
copyrightSpringer Science+Business Media New York 1995
The information of publication is updating

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t groups to express views on similar topics. Perhaps, however, the most surprising feature of the workshop was the lack of controversy regarding the role of Epstein-Barr virus in Hodgkin‘s disease, an association that was treated with great scepticism at the beginning of the decade. The first three chapters, 978-1-4613-8005-4978-1-4613-0339-8
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https://doi.org/10.1007/978-3-319-54879-1e individuals are placed in culture, the few virus-infected B-cells that are present regularly give rise to spontaneous outgrowth of EBV-transformed LCLs provided that immune T-cells are either removed or inhibited by addition of cyclosporin A to the culture (Rickinson .., 1984). This phenomenon hig
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Development Administration in the Caribbeanominant type the neoplastic cells express B-cell markers (Coles ..., 1988; Kadin .., 1988), whereas in other subtypes B and/or T-cell markers can be found on H-RS cells in a variable proportion of cases (Oka .., 1988; Kadin .., 1988; Bjarni ..., 1989; Cibull .., 1989).
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The Theory of Economic Development,ar .., 1991; Levine .., 1992a) and virological (Torelli .., 1991; Gompels .., 1993) studies. This report will summarise the data suggesting an aetiological association, review relevant studies to place these findings in perspective, and discuss possible approaches to the identification of other cand
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https://doi.org/10.1007/978-3-319-47925-5ogressively from 3 of 15 HD biopsy specimens transplanted into SCID mice. The tumours were derived from EBV-positive bystander cells. In 5/6 tumours examined cytogenetically, chromosomal aberrations were detected. We conclude that these bystander cells might differ from normal lymphocytes and possib
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