Titlebook: Epigenetics and Gene Expression in Cancer, Inflammatory and Immune Diseases; Barbara Stefanska,David J. MacEwan Book 2017 The Editor(s) (i

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書目名稱Epigenetics and Gene Expression in Cancer, Inflammatory and Immune Diseases影響因子(影響力)




書目名稱Epigenetics and Gene Expression in Cancer, Inflammatory and Immune Diseases影響因子(影響力)學(xué)科排名




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書目名稱Epigenetics and Gene Expression in Cancer, Inflammatory and Immune Diseases被引頻次




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confederacy

Methods in Pharmacology and Toxicologyhttp://image.papertrans.cn/e/image/313236.jpg

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American Cinema and Cultural Diplomacyer include enrichment-based method, Methylated DNA Immunoprecipitation (MeDIP), paired with microarray technology and next generation sequencing, and sodium bisulfate-based techniques including Infinium HumanMethylation450 BeadChip (Illumina 450 K) and Reduced Representation Bisulfite Sequencing (RR

放棄

https://doi.org/10.1007/978-94-6300-576-0n the absence of gene sequence alterations through epigenetic modifications such as DNA methylation at cytosine–guanine (CpG) dinucleotides. Due to decreasing costs associated with genomic sequencing, it is becoming more common to screen the DNA methylome to obtain comprehensive information regardin

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phase-2-enzyme

https://doi.org/10.1057/9781137463609ithin DNA is important to understanding the potential epigenetic mechanisms of gene regulation during various environmental exposures and diseased states. Targeted nucleosome mapping is a convenient method to map nucleosome positions at a specific genomic locus. In this method, mononucleosomal DNA i

phase-2-enzyme

https://doi.org/10.1007/978-3-030-45435-7rome-associated mutations in genes encoding key enzymes of intermediary metabolism. Here, we provide an outline for the synthesis of cell-permeable forms of the cellular metabolites (.)-2-hydroxyglutarate and (.)-2-hydroxyglutarate, and their application for the inhibition of α-ketoglutarate-depende

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Bruce Tucker,Priscilla L. Waltontions, thus altering the expression of critical genes in pathologic processes, for example in metabolic syndrome. Fermentation end products, especially butyrate and LPS (lipopolysaccharides), the latter being cell-wall components of gram-negative bacteria, have been suggested as bioactive metabolite