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Titlebook: Endocrinology of Cardiovascular Function; Ellis R. Levin,Jerry L. Nadler Book 1998 Springer Science+Business Media New York 1998 Angiotens

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發(fā)表于 2025-3-23 10:40:47 | 只看該作者
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發(fā)表于 2025-3-23 15:45:17 | 只看該作者
Insulin-Like Growth Factor I and the Heart growth via its endocrine, paracrine and autocrine actons. Accumulating evidence has demonstrated involvement of IGF-I in the regulation of myocardial growth and function in both normal and diseased hearts, indicating that IGF-I signaling is part of the signaling pathways controlling myocardial grow
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發(fā)表于 2025-3-23 20:52:32 | 只看該作者
https://doi.org/10.1007/978-981-10-5029-9ce from these and other studies suggests that estrogen may be cardioprotective through a combination of mechanisms. For example, estrogen replacement therapy results in decreased plasma low-density lipoprotein (LDL) and increased high-density lipoprotein (HDL) concentrations (.-.). Other benefits of
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發(fā)表于 2025-3-24 13:14:32 | 只看該作者
1566-0729 ells. Other `endocrine‘ hormones, such as PTHRP andadrenomedullin, also modulate cardiovascular and renovascular dynamicstates, suggesting new roles for these p978-1-4613-7547-0978-1-4615-5569-8Series ISSN 1566-0729
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發(fā)表于 2025-3-24 17:08:10 | 只看該作者
Book 1998have been found to participate in preventing the development ofatherosclerosis in women, acting through novel mechanisms on targetvascular cells. Other `endocrine‘ hormones, such as PTHRP andadrenomedullin, also modulate cardiovascular and renovascular dynamicstates, suggesting new roles for these p
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發(fā)表于 2025-3-24 19:33:16 | 只看該作者
Effects of Sex Steroids in Vascular Injuryce from these and other studies suggests that estrogen may be cardioprotective through a combination of mechanisms. For example, estrogen replacement therapy results in decreased plasma low-density lipoprotein (LDL) and increased high-density lipoprotein (HDL) concentrations (.-.). Other benefits of
20#
發(fā)表于 2025-3-24 23:42:24 | 只看該作者
Angiotensin II, Growth Factors and the Lipoxygenase Pathwaychapter also examines the effects of hyperglycemia on the regulation and function of the lipoxygenase pathway, in order to evaluate potential mechanisms for the accelerated cardiovascular disease observed in diabetics.
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