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Titlebook: Dendritic Cells and Virus Infection; Alexander Steinkasserer Book 2003 The Editor(s) (if applicable) and The Author(s), under exclusive li

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發(fā)表于 2025-3-21 19:31:51 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Dendritic Cells and Virus Infection
編輯Alexander Steinkasserer
視頻videohttp://file.papertrans.cn/266/265513/265513.mp4
叢書名稱Current Topics in Microbiology and Immunology
圖書封面Titlebook: Dendritic Cells and Virus Infection;  Alexander Steinkasserer Book 2003 The Editor(s) (if applicable) and The Author(s), under exclusive li
描述Dendritic cells are vital to induce potent anti-viral immune responses. It will become clear to the reader that dendritic cells often play a dual role during viral infections. On the one hand they are able to mount potent antiviral immune responses, and on the other hand several viruses, including HIV-1, use DC as a vector to be transferred from the periphery to the lymph nodes where they infect their prime target.
出版日期Book 2003
關(guān)鍵詞Antigen; Epstein-Barr Virus; HIV; Herpes Simplex Virus-1; Immunodeficiency Virus; cell; cells; dendritic ce
版次1
doihttps://doi.org/10.1007/978-3-662-06508-2
isbn_softcover978-3-642-07926-9
isbn_ebook978-3-662-06508-2Series ISSN 0070-217X Series E-ISSN 2196-9965
issn_series 0070-217X
copyrightThe Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-Verlag GmbH, DE
The information of publication is updating

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發(fā)表于 2025-3-21 21:23:18 | 只看該作者
Nontraditional Options for Disseminationion with its immunological ligand ICAM-3, recent evidence demonstrates that glycosylation of gp120 is not necessary for its interaction with DC-SIGN. Moreover, mutational analysis demonstrates that the HIV-1 gp120 binding site in DC-SIGN is different from that of ICAM-3..Besides its role in DC-media
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發(fā)表于 2025-3-22 03:23:05 | 只看該作者
Susan Flynn,Lucas Evans,Henry Sessangag protective EBV immunity and that the EBV nuclear antigen 1 (EBNA1)-specific CD4. T cell response probably plays a role in resistance against all types of EBV-associated malignancies in healthy carriers. This implies that EBNA1 targeting to dendritic cells should be a component of vaccine and immun
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發(fā)表于 2025-3-22 06:51:16 | 只看該作者
Jun Li,Peter Reiher,Gerald J. Popek instrumental in MV-induced immunosuppression. (1) T cell depletion could be brought about by DC-T cell fusion or TRAIL-mediated induction of apoptosis. (2) Inhibition of stimulated IL-12 production from MV-infected DCs might affect T cell responses in qualitative terms in favouring Th2 and suppress
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發(fā)表于 2025-3-22 09:16:55 | 只看該作者
Dissemination of Cartographic Knowledgealso targets DCs: Progress in understanding the immunobiology of MV-infected DCs that could account for MV-induced immunosuppression observed in vivo is presented and their potential role in lymphopenia is underlined. In conclusion, future research directions are proposed.
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發(fā)表于 2025-3-22 18:21:41 | 只看該作者
Dissemination of Cartographic Knowledgenfection in lysosomal compartments. This CD83 degradation coincided with a clearly reduced T cell stimulation representing an additional new escape strategy. The functional role and the importance of CD83 are discussed in detail.
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發(fā)表于 2025-3-22 22:49:30 | 只看該作者
https://doi.org/10.1007/978-3-031-56019-4microbial immune responses. Here, we discuss recent data on induction of antiviral immunity with various DC-vaccination approaches and outline future directions for the development of specific antigen targeting to DCs.
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發(fā)表于 2025-3-23 08:57:55 | 只看該作者
Disagreement and Religious Relevance epitopes. Whole tumour cell preparations are difficult to standardise, and they depend on the availability of tumour cells. Thus the utilisation of viral vectors genetically modified to express TAA for the ex vivo transduction of DCs is an attractive alternative to achieve a MHC I- and MHC II-restr
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