Titlebook: Cytochrome Oxidase in Neuronal Metabolism and Alzheimer’s Disease; F. Gonzalez-Lima Book 1998 Springer Science+Business Media New York 199

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Agnosia

Behavioral, Electrophysiological, and Biochemical Consequences of Chronic Cytochrome Oxidase Inhibits have shown that C.O. activity is reduced in both blood platelets and in the brains of patients with Alzheimer’s disease (A.D.), suggesting that a decline in the efficiency of this enzyme could impair cognition. We explored this idea by chronically treating rats with sodium azide, using doses that

Hypomania

Cytochrome Oxidaser predicting potential neurodegeneration, particularly following chronic brain hypoperfusion. This proposal is based on a series of experiments in rats subjected to mild chronic brain hypoperfusion and tested at determined time points for regional cytochrome oxidase activity, visuo-spatial memory, r

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Cytochrome Oxidase Atlas of Rat Brainections stained with cresyl violet. We used a fresh-frozen brain, without perfusion-fixation, from an adult hooded male rat. Bregma coordinates and brain structures are identified for each of fourteen Bregma levels in the coronal plane.

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A Guide to Use of the Collection,ew functional neuroimaging technique for examining baseline changes in activity which have taken place over a period of time, as opposed to evoked activity alone. Cytochrome oxidase is the pivotal mitochondrial oxidative enzyme for energy production in cells. The amount of this enzyme is tightly cou