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Titlebook: Coronary Circulation in Physiological and Pathophysiological States; Motoomi Nakamura,Paul M. Vanhoutte Book 1991 Springer-Verlag Tokyo 19

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樓主: Wilder
31#
發(fā)表于 2025-3-26 21:46:53 | 只看該作者
Effects of Atherosclerosis and Regression of Lesions, pathophysiology of vasospasm. The second goal is to determine whether vasomotor abnormalities of atherosclerotic arteries are corrected by regression of lesions..The experimental model is cynomolgus monkeys which develop atherosclerotic lesions that closely resemble those occurring in humans. Monke
32#
發(fā)表于 2025-3-27 04:59:35 | 只看該作者
Vascular Regulation in Atherosclerosis,cular channels. Current evidence indicates that the arteriosclerotic circulation undergoes several alterations that limit acute and chronic hyperemic responses. Macrovascular occlusive disease (atheroma, thrombosis) is not the only factor that may limit flow. Vasomotor tone, the sum of dilating and
33#
發(fā)表于 2025-3-27 08:05:08 | 只看該作者
34#
發(fā)表于 2025-3-27 11:05:55 | 只看該作者
35#
發(fā)表于 2025-3-27 15:55:38 | 只看該作者
36#
發(fā)表于 2025-3-27 21:42:54 | 只看該作者
Installing Third-Party Libraries,siological data on α- and β-adrenergic receptors were correlated with ligand binding studies from sarcolemmal membranes of calf large coronary arteries. In the conscious animal, selective α.- + α.- and β. - + β.-adrenergic agonists were examined in the presence and absence of selective α.- + α.-, an
37#
發(fā)表于 2025-3-28 01:52:53 | 只看該作者
38#
發(fā)表于 2025-3-28 03:43:00 | 只看該作者
39#
發(fā)表于 2025-3-28 10:19:20 | 只看該作者
Project 5: Remote Control Hardware,ich most likely is nitric oxide, or a nitrosoderivative releasing nitric oxide, derived from 1-arginine), and endothelium-derived hyperpolarizing factor (EDHF, which possibly is a labile metabolite of arachidonic acid formed through the P-450 pathway). Possible endothelium-derived contracting factor
40#
發(fā)表于 2025-3-28 11:49:42 | 只看該作者
What Is the Internet of Things?, pathophysiology of vasospasm. The second goal is to determine whether vasomotor abnormalities of atherosclerotic arteries are corrected by regression of lesions..The experimental model is cynomolgus monkeys which develop atherosclerotic lesions that closely resemble those occurring in humans. Monke
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