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Titlebook: Coronary Circulation in Physiological and Pathophysiological States; Motoomi Nakamura,Paul M. Vanhoutte Book 1991 Springer-Verlag Tokyo 19

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書目名稱Coronary Circulation in Physiological and Pathophysiological States
編輯Motoomi Nakamura,Paul M. Vanhoutte
視頻videohttp://file.papertrans.cn/239/238321/238321.mp4
圖書封面Titlebook: Coronary Circulation in Physiological and Pathophysiological States;  Motoomi Nakamura,Paul M. Vanhoutte Book 1991 Springer-Verlag Tokyo 19
描述MOTOOMI NAKAMURA As we approach the 21st century, ischemic heart disease is the major cause of death in most of the developed nations of the world. Since the 1970s, much effort and expense have led to designs of coronary thrombolytic therapy, percutaneous coronary angioplasty (PTCA), coronary artery bypass grafting, heart transplantation, automatic defibrillators, as well as to the formation of beta blockers and com- pounds which block the calcium channel. Socio-educational programs directed at exercise, diet, instruction in the risk factors of smoking, hyperlipidemia and hypertension have contributed to the decrease in the rate of morbidity and mortality of patients with ischemic heart disease. However, the first clinical event of ischemic heart disease, the so-called "heart attack" and sudden cardiac death continues to present problems, as the mechanisms involved in these events are poorly understood. It has long been thought that ischemic heart disease is the sequence of an organic fixed atherosclerotic obstruction of the epicardial coronary arteries and the role of coronary vasomotion has been given much less attention. Recent clinical and laboratory animal studies revealed tha
出版日期Book 1991
關(guān)鍵詞Herz-Kreislauf-System; angiogenesis; atherosclerosis; cell; cell biology; cells; circulation; endothelium; h
版次1
doihttps://doi.org/10.1007/978-4-431-68108-3
isbn_softcover978-4-431-68110-6
isbn_ebook978-4-431-68108-3
copyrightSpringer-Verlag Tokyo 1991
The information of publication is updating

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world. Since the 1970s, much effort and expense have led to designs of coronary thrombolytic therapy, percutaneous coronary angioplasty (PTCA), coronary artery bypass grafting, heart transplantation, automatic defibrillators, as well as to the formation of beta blockers and com- pounds which block
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Containerizing existing applications,e biophysical features of vascular smooth muscle cell membranes are reviewed in relation to coronary vasospasm, as well as the actions of some clinical drugs—nitro-(nitroso-, nitrite-) compounds, β-adrenoceptor blockers, Ca antagonists, and K channel openers—on ionic channels in vascular smooth muscle cells.
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Installing Third-Party Libraries,esponding blocker for the other subtype. Ligand binding studies demonstrated the presence of both subtypes of α- and β-adrenergic receptors, with a predominance of the β. and α.-adrenergic receptor subtypes. Thus, large coronary arteries of the calf contain both α.- + α.- and β.- + β.-adrenergic rec
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Project 5: Remote Control Hardware,tacyclin plays a key role in the protective role of the endothelium against vasospasm and unwanted coagulation of blood. Indeed, thrombin and aggregating platelets are potent stimuli for the release of endothelium-derived relaxing factor. The platelet products responsible are the adenine nucleotides
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