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Titlebook: Clinical Pharmacology of Antianginal Drugs; Ulrich Abshagen (Leiter Produktentwicklung Therape Book 1985 Springer-Verlag Berlin Heidelberg

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發(fā)表于 2025-3-21 16:16:28 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Clinical Pharmacology of Antianginal Drugs
編輯Ulrich Abshagen (Leiter Produktentwicklung Therape
視頻videohttp://file.papertrans.cn/229/228170/228170.mp4
叢書名稱Handbook of Experimental Pharmacology
圖書封面Titlebook: Clinical Pharmacology of Antianginal Drugs;  Ulrich Abshagen (Leiter Produktentwicklung Therape Book 1985 Springer-Verlag Berlin Heidelberg
描述When I was asked some years ago by the editors of the Handbook of Experimental Pharmacology to edit a new volume on Antianginal Drugs, I agreed on the condition that, in accordance with my scientific background, primary emphasis be given to clinical pharmacology and therapeutics. It soon turned out that, due to rapid developments in this field, nothing of the previous volume on Antianginal Drugs by Charlier (Vol. 31, 1971) could be retained apart from its basic idea of devoting considerable space to methodology. Since editors must operate within certain limits, I had to abstain from dealing with acute myocardial infarction in detail despite the well-known overlap between unstable angina, the preinfarction syndrome, and acute myocardial infarction. It was only possible for acute myocardial infarction and the concept of reduction of infarct size to be briefly discussed within the chapter on pathophysiology of acute coronary insufficiency. The chapter on invasive methods provided an opportunity to touch on new approaches to early intervention in acute myocardial infarction. Here, intracoronary streptokinase therapy and PTCA are considered, again with attention to the overlap between m
出版日期Book 1985
關鍵詞Drugs; PTCA; pathophysiology; pharmacology; physiology; research
版次1
doihttps://doi.org/10.1007/978-3-642-69524-7
isbn_softcover978-3-642-69526-1
isbn_ebook978-3-642-69524-7Series ISSN 0171-2004 Series E-ISSN 1865-0325
issn_series 0171-2004
copyrightSpringer-Verlag Berlin Heidelberg 1985
The information of publication is updating

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https://doi.org/10.1007/978-3-642-69524-7Drugs; PTCA; pathophysiology; pharmacology; physiology; research
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,Sachlichkeitsp?dagogik und Pflichtethik,s our generation did between the 1950s and 1980s. Whereas, in the western hemisphere, infectious diseases came under control and, subsequently, rheumatic heart disease and syphilitic cardiovascular complications were almost eradicated, rapidly increasing “diseases of civilization” accounted for mill
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https://doi.org/10.1007/978-3-658-36683-4athological disorder. It follows that throughout the ages the pharmacodynamic principles of action have been directly dependent on the prevailing concepts of pathology and developed in close conjunction with advances in this field. The clinical pharmacological testing of preparations on patients on
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Everhard Holtmann,Bernhard Bollkers observed that acute, reversible changes in the ECG occurred during spontaneous attacks of angina pectoris. In 1928, . and . showed that similar changes in the ECG occurred when angina pectoris was precipitated by exercise. . (1932) later demonstrated the same exerciseinduced abnormalities in ov
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https://doi.org/10.1007/978-3-642-67660-4of nonbenzenoid aromatic compounds that posses a so-called mesoionic structure. Such a compound has never been introduced into therapy despite known multiple pharmacologic properties of different sydnones and sydnonimines (A. 1967; D. and D. 1962a-c; K. and R. 1967; O. et al. 1965). The drug was syn
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