Titlebook: Cerebral Ischemia; Molecular and Cellul Wolfgang Walz Book 1999 Springer Science+Business Media New York 1999 brain.brain injury.cell.cereb

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Video Imaging and Photodocumentationes are two examples of such pleiotropic signaling moieties released during inflammation that can have diverse effects on neurons under pathophysiological conditions. Both tumor necrosis factor α (TNFa) and transforming growth factor β(TGFβ) can protect neurons from metabolic, excitotoxic, and oxidat

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periodontitis

Cell Swelling in Cerebral Ischemiavement of the permeant anions down their concentration gradients into the cells. However, this membrane potential will be reduced (less negative) when the membrane is also permeable to the cations whose equilibrium potential is zero. Thus, this situation is not stable; there will always be movement

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Oxygen Radicalsmonstrated that about 2–5% of the electron nitric oxide (NO) flow in isolated brain mitochondria produces superoxide (O..) and hydrogen (H.O.) (Boveris and Chance, 1973). These constantly produced oxygen radicals are scavenged respectively by superoxide dismutases (SODs), glutathione peroxidases (GS

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Reprogramming of Gene Expression After Ischemiall demonstrate the influence of postischemic protein synthesis deficits on mRNA translation. Within this framework, consideration will then be given to other ischemia-induced gene products, including other transcription factors and stress proteins, as well as neurotrophic factors, components of oxid

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Book 1999nsuming processes is mainly caused by the active transport of ions, which is necessary to compensate for the transmembrane ion currents that are part ofthe complex signaling processes in the brain. Ninety-five percent ofthe brain‘s ATP is derived from mitochondrial oxidative phosphorylation. Since t

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Surgical Anatomy of the Eyelid,damaged core areas into the surrounding penumbra in focal ischemia. Recently, it has also been shown that SD waves in healthy tissue can have a neuroprotective effect. For these reasons, there is renewed interest in SD and it’s propagation mechanisms.

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https://doi.org/10.1007/978-3-662-22265-2nt scientific investigations have revealed the detailed features of such classical and modern concepts of ischemic neuronal death. The present chapter describes and compares characteristic classification of two major types of ischemic neuronal death, and introduces a current idea synthesizing the two mechanisms of cell death.