Titlebook: Cerebral Ischemia; Molecular and Cellul Wolfgang Walz Book 1999 Springer Science+Business Media New York 1999 brain.brain injury.cell.cereb

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書目名稱Cerebral Ischemia影響因子(影響力)




書目名稱Cerebral Ischemia影響因子(影響力)學(xué)科排名




書目名稱Cerebral Ischemia網(wǎng)絡(luò)公開度




書目名稱Cerebral Ischemia網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Cerebral Ischemia被引頻次




書目名稱Cerebral Ischemia被引頻次學(xué)科排名




書目名稱Cerebral Ischemia年度引用




書目名稱Cerebral Ischemia年度引用學(xué)科排名




書目名稱Cerebral Ischemia讀者反饋




書目名稱Cerebral Ischemia讀者反饋學(xué)科排名

清澈

browbeat

978-1-4757-4735-5Springer Science+Business Media New York 1999

JUST

Springer Series in Optical Sciencesre a result of cerebral ischemia (Mohr et al., 1978). Global cerebral ischemia involves the entire brain and occurs during cardiac arrest or severe systemic hypotension. Focal cerebral ischemia affects restricted brain regions and occurs in a wide variety of clinical settings but is most commonly a

Cougar

vascular

https://doi.org/10.1007/978-3-662-22251-5ules surrounded by a semipermeable lipid membrane. This is speculated as being the prototypic cell—a spontaneously forming phospholipid bilayer membrane enclosing enough protein and nucleic acids to form a nonequilibrium, self-replicating entity (Tosteson, 1963b). Donnan forces arise because of the

vascular

A. Cuschieri,L. K. Nathanson,S. M. Shimichemic cell death has greatly improved. An ion that has assumed major importance in causing death of the ischemic cell is Cat., hence, various efforts have been directed to controlling the intracellular concentration of this ion. This “classical” Cat. hypothesis of cell death has evolved to the more

不透明

Flatter

Acetaminophen

Molecular Biology Intelligence Unith responses in brain differ considerably after global and focal ischemia and are strongly dependent on the duration and severity of blood flow deficits. This review initially focuses on inducible transcription factors encoded by cellular immediate-early genes (cIEGs) of the . multigene families, and