Titlebook: Cavernomas of the CNS; Basic Science to Cli Ond?ej Bradá?,Vladimír Bene? Book 2020 Springer Nature Switzerland AG 2020 cavernous malformati

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Introduction

Molecular Biology of CCM,nd structurally irregular capillaries which exhibit dilated, thin and leaky walls. CCMs can occur either sporadically or as a familial autosomal disorder caused by a germline mutation with variable clinical and neuroradiological penetrance. Despite similar neurological manifestations, prevalence of

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Presentation,age [ICH, 25%], and non-hemorrhagic focal neurologic deficits [NH-FNDs, 25%]. A significant number of CCM patients [20%–50%] is asymptomatic. CCM related symptoms can be attributed to intralesional or extralesional hemorrhage, mass effect, or other mechanisms including perilesional hemosiderin depos

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Hemispherical Cavernomas in Non-Eloquent and Eloquent Areas,le the location of CCMs is quite variable, 70%–80% of CCMs have a supratentorial origin. With the increasing availability of MR imaging, the frequency of diagnosis of CM has risen significantly..Treatment options for patients with CCM include observation, microsurgical resection, and at times stereo

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Cavernoma-Related Epilepsy,urologists’ ability to consult patients regarding conservative or operative treatment approaches and the natural history of the disease. We add nuances from our own clinical experience and give example of typical cases.

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Surgery of Deep-Seated Cavernous Malformations,n three factors: The natural history of the lesions, benefits and risks of treating the lesions, and the surgical accessibility. Over the last few decades, surgical resection of deep-seated CMs has become more common and the use of modern techniques has yielded improved results, although the number