Titlebook: Cardiovascular Specific Gene Expression; Pieter A. Doevendans,Robert S. Reneman,Marc Bilsen Book 1999 Springer Science+Business Media Dord

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Optimizing Electromagnetic Devicesand in this way conduct the cardiac action potential. This results in synchronized activation of the cardiac cells and allows coordinated contraction of the heart. Gap junction channels (for a review see [1]) consist of two hemi-channels and these in turn are made up of 6 protein subunits, connexins

Obsequious

Variational Finite Element Methodtern must be established of time-dependent changes in cellular permeability to maintain the cardiac cycle that underlies normal cardiac function. Impulses, which originate in the sinoatrial (SA) node, are conducted by the myocardium throughout the atria until they converge at the atrioventricular (A

Awning

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Surface Charge Simulation Method (SSM)of families with cardiac diseases like hypertrophic cardiomyopathy and dilated cardiomyopathy (DCM). Although four chromosomal loci are associated with the inherited form of DCM, the genes involved are still unknown [1]. In most patients with dilated cardiomyopathy inheritance appears less important and the etiology remains obscure.

百靈鳥

Cardiology Approaching the Year 2000. A Clinician’s Look at Molecular Cardiologyaths were caused by cardiovascular disease and this is expected to rise to 36% in 2020. Incidence and ability to lower the number of cardiovascular deaths varies considerably from country to country. As shown by Sans et al. [2] standardized mortality from ischemic heart disease may vary in Europe fr

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河潭

A Cardiac-Specific Troponin I Promoter. Distinctive Patterns of Regulation in Cultured Fetal Cardiom SP1 and TEF-1, and genes coding for specific transcription factors: MADS-box transcription factor genes, such as the MEF2 genes which are also involved in the specification of skeletal and smooth muscle, homeobox genes, such as Nkx2.5, zinc-finger genes of the GATA family, such as GATA 4–6, and bHL

Myelin

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