Titlebook: Captopril and Hypertension; David B. Case,Edmund H. Sonnenblick,John H. Laragh Book 1980 Springer Science+Business Media New York 1980 blo

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Chameleon

https://doi.org/10.1057/9780230510869st be administered parenterally, which makes chronic blockade of the system practically impossible. Furthermore, saralasin, a competitive inhibitor of the active hormone angiotensin II, has the disadvantage of inherent agonistic properties.. On the other hand, because angiotensin-converting enzyme i

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New Approaches to Religion and Powerunity to determine and to quantify the extent to which the renin-angiotensin system participates in the maintenance of hypertensive states. High levels of plasma renin activity relative to the state of sodium balance have been documented in patients with malignant, surgically remediable renovascular

改變

Gentry

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Memorial

https://doi.org/10.1007/978-0-230-23366-9-converting enzyme (ACE) [E.C. 3.4.15.1] has also been designated as converting enzyme (CE), peptidyldipeptide carboxy hydrolase, kininase II, or “bradykininase.” Hence, in vivo inhibition of ACE will reduce the pressor activity of AI, but not that of AII, and augment the vasodepressor activity of bradykinin (BK).

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The Challenge for , Organizations,bin et al. in other chapters in this volume. This chapter will delineate the results of a number of studies that characterized the acute and subacute toxicities of SQ 14,225 in laboratory animals. Some metabolic data from preliminary studies in rats and dogs are also included.

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