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Titlebook: Brain and Heart Infarct II; K. J. Zülch,W. Kaufmann,V. Hossmann Conference proceedings 1979 Springer-Verlag Berlin Heidelberg 1979 brain.c

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發(fā)表于 2025-3-21 17:16:10 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Brain and Heart Infarct II
影響因子2023K. J. Zülch,W. Kaufmann,V. Hossmann
視頻videohttp://file.papertrans.cn/191/190261/190261.mp4
圖書封面Titlebook: Brain and Heart Infarct II;  K. J. Zülch,W. Kaufmann,V. Hossmann Conference proceedings 1979 Springer-Verlag Berlin Heidelberg 1979 brain.c
影響因子Dr. Kaufmann and I wish to welcome you here in Schloss Auel to this second symposium on brain and heart infarct. We have chosen this lovely castle because I am always dissatisfied with meetings in great hotels in cities, where we rush to and from the meeting rooms. I had such an experience overseas just 3 weeks ago from 8 o‘clock in the morning until 10 o‘clock at night. We want you to feel some of the wonderful atmosphere of the Bergisches La~d. Moreover, we wanted like you to enjoy a bit of the spirit of the Rhein valley and the 2000-year-old city of Cologne, even at the expense of losing a few hours of discus- sion; though this may be a quantitative loss, we feel sure that the environment of this city will stimulate our discussions. Two years ago we gathered here in Schloss Auel for the first time, attempting to conduct a discussion between neurologists and cardiologists on the similarities and dissimilarities of circulatory disorders of the brain and heart. We are happy to continue this discussi?n in the days to come.
Pindex Conference proceedings 1979
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Epidemiology of Coronary Heart Disease,d with the investigation of the factors that determine its occurrence and natural history. Coronary heart disease (also referred to as ischemic heart disease in the International Classification of Diseases) is the leading cause of death in affluent countries. From being a medical rarity prior to the
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Local Factors Contributing to the Pathogenesis of Atherosclerosis,and from individual to individual. However, quite apart from the species and individual susceptibility, it has long been recognized that local tissue factors play a role in the pathogenesis of this disease (1). Many authors speak glibly of hypothetic injury to the vessel wall, but the only site in t
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Quantitation of Coronary Arterial Luminal Narrowing in Coronary Heart Disease,(right, left anterior descending, and left circumflex) coronary arteries in patients with fatal acute myocardial infarction. Roberts and Buja (3) in 1972 by histologic study of cross sections of 5-mm long segments of the three major coronary arteries also found among patients with fatal acute myocar
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Predilection of Cerebral Atherosclerotic Stenosis: a Morphologic and Radiologic Demonstration,nce, by the World Federation of Neurology (7) on the basis of which it has directed corresponding world-wide research programs on the epidemiology of cerebrovascular disease (2, 6). We have made similar studies in our institute by measuring the lumina of the arterial stenoses (1, 9; Figs. 10–14).
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Development and Regression of Experimental Local Carotid Atheromatosis in Rabbits,of the vessels that have to undergo massive mechanical stimuli (e.g., orifices of branching arteries) and that these regions become foci of predilection for lipid deposition if the lipid concentration of the blood is high. To produce such plaques artificially in animal experiments, it is necessary t
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Turbulence Generation in Stenotic Blood Vessel Models,aques to develop at certain discrete sites at which disturbances of blood flow are apt to take place prompted many investigators to speculate that the local hemodynamic environment is an active agent in the pathogenesis and proliferation of atherosclerosis (7). Intravascular hemolysis and thrombus f
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Hemorheology and Ischemia, wall with which blood or its constituents come into direct contact. Ischemia has been defined as a local anemia in a part of the body due to either sudden or gradual cutting-off or obstruction of the arterial blood supply to the affected area. The usual causes for sudden obstruction in pathologic c
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Ion-Selective Electrodes in Cardiac Ischemia,, 6, 8, 10). Until recently, changes in the extracellular ion concentrations could be investigated only by measuring arteriocoronary venous differences (1, 8). Since considerable ion gradients between venous blood and extracellular space were observed during impeded blood supply (3, 5, 6), extracell
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