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Titlebook: Brain Ischemia; Eugene Gusev,Veronica I. Skvortsova Book 2003 Springer Science+Business Media New York 2003 brain.death.depression.glutama

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樓主: Mosquito
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發(fā)表于 2025-3-23 13:20:19 | 只看該作者
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發(fā)表于 2025-3-23 16:10:29 | 只看該作者
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發(fā)表于 2025-3-23 18:52:51 | 只看該作者
ent blood is delivered to the brain. Many physiological processes occurring in the brain critically depend on the state of its energy metabolism. The state of brain energy metabolism in turn depends on the delivery of oxygen and glucose to the brain via the bloodstream. Although it comprises only 2%
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發(fā)表于 2025-3-24 01:12:51 | 只看該作者
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發(fā)表于 2025-3-24 06:17:11 | 只看該作者
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發(fā)表于 2025-3-24 08:48:46 | 只看該作者
Metabolic Acidosis and Ischemic DamageH. concentration in early stages of ischemia plays a compensatory and adaptive role as it promotes the improvement of perfusion in the penumbral area [.]. Significant elevation of lactate level within the first hours of ischemic stroke leads to decrease in pH to 6.4–6.7 [.] and appears to be an unfavorable prognostic sign [., ., ., .].
17#
發(fā)表于 2025-3-24 10:40:56 | 只看該作者
Reparative Therapytive processes, plasticity of brain tissue, and the formation of new associative pathways, along with neuroprotection aimed at prevention of further brain damage and resulting enlargement of infarction volume.
18#
發(fā)表于 2025-3-24 15:02:02 | 只看該作者
A Brief Introduction on Kernels, the penumbra determine the final size of the brain infarction formed by the 3rd to 5th days after stroke onset. These changes must be studied to establish the basis for developing effective neuroprotective therapeutic approaches [., ., ., ., ., .].
19#
發(fā)表于 2025-3-24 21:28:41 | 只看該作者
https://doi.org/10.1007/978-3-658-40237-2ils the risk of systemic dysfunction when any one component of the whole system is affected. Such disorders can be defined as under-regulation pathology, the pathogenesis of which may be connected with primarily nervous or endocrine and/or immune mechanisms [.].
20#
發(fā)表于 2025-3-24 23:20:08 | 只看該作者
Fuwei Li,Lifeng Lai,Shuguang Cuihat to a considerable extent determine the features of the course of disease [., ., .]. This response is mediated by the sympathoadrenal, hypothalamic-pituitary-adrenal, renin-angiotensin, and thyroid hormonal axes [.].
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