Titlebook: Brain Ischemia; Eugene Gusev,Veronica I. Skvortsova Book 2003 Springer Science+Business Media New York 2003 brain.death.depression.glutama

Mosquito

書目名稱Brain Ischemia影響因子(影響力)




書目名稱Brain Ischemia影響因子(影響力)學(xué)科排名




書目名稱Brain Ischemia網(wǎng)絡(luò)公開(kāi)度




書目名稱Brain Ischemia網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書目名稱Brain Ischemia被引頻次




書目名稱Brain Ischemia被引頻次學(xué)科排名




書目名稱Brain Ischemia年度引用




書目名稱Brain Ischemia年度引用學(xué)科排名




書目名稱Brain Ischemia讀者反饋




書目名稱Brain Ischemia讀者反饋學(xué)科排名

cathartic

Energy Failure Induced by Brain Ischemia a substrate for cytochrome oxidase, the terminal enzyme of the respiratory chain. The resulting state of hypoxia is the consequence of a complicated multi-stage process involving sequential changes in the properties of the mitochondrial enzyme complexes [.].

Nebulous

Metabolic Acidosis and Ischemic Damagey activation of anaerobic glycolysis and to increased production of lactate and H. that causes the development of lactic acidosis. Modest increase in H. concentration in early stages of ischemia plays a compensatory and adaptive role as it promotes the improvement of perfusion in the penumbral area

Fecal-Impaction

Thrombolysis

Autoimmune Mechanisms of Trophic Dysfunction and Ischemic Brain Damagethey interact via mutual regulation involving neuromediators, neuropeptides, cytokines, trophic factors, and hormones and the corresponding receptor systems [.]. Mutual regulation of the nervous, endocrine, and immune systems determines the reliability of their common functioning. However, this enta

我要沮喪

Reaction of the Stress-Mediating Endocrine System in Response to Acute Brain Ischemiastem caused by the expression of immediate-early genes—by the synthesis of heat-shock (or stress) proteins (HSP) and cytokines, which are immune mediators [., ., ., .] (see Chapters 7 and 8; Fig. 8.1). The stress-mediating response provides the metabolie basis for compensatory and adaptive changes t

尾隨

Condyle

Reparative Therapyin tissue. Thus, from the first hours after stroke onset it is necessary to proceed with treatment aimed at the improvement of reparative and regenerative processes, plasticity of brain tissue, and the formation of new associative pathways, along with neuroprotection aimed at prevention of further b

忙碌

Vimal K. Shrivastava,Monoj K. Pradhanant to mention not only its therapeutic, but also preventive value. Correction of delayed consequences of ischemia can lead to retardation of vascular encephalopathy development in the post-stroke period.

Hyperopia

Secondary Neuroprotectionant to mention not only its therapeutic, but also preventive value. Correction of delayed consequences of ischemia can lead to retardation of vascular encephalopathy development in the post-stroke period.