Titlebook: Brain Damage and Repair; From Molecular Resea T. Herdegen,J. Delgado-García Book 2004 Springer Science+Business Media B.V. 2004

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書目名稱Brain Damage and Repair影響因子(影響力)




書目名稱Brain Damage and Repair影響因子(影響力)學科排名




書目名稱Brain Damage and Repair網絡公開度




書目名稱Brain Damage and Repair網絡公開度學科排名




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書目名稱Brain Damage and Repair被引頻次學科排名




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defile

Amenable

infinite

The Organelles II: Endoplasmic Reticulum and its Overloadfunctioning is impaired in multiple ways. These include disturbances of ER calcium homeostasis, impairment of ERAD and UPR, and insufficient proteasome functioning which triggers secondary ER dysfunction. Therapeutic interventions designed to suppress the pathological process culminating in neuronal

鞏固

Transcriptional Dysfunctions as Pathogenic Mechanism of Neurodegenerative Diseases protein diversity and determine the subcellular location, molecular interactions, or function of proteins. Neuron-specific proteins involved in RNA splicing and metabolism are affected in several neurological disorders. Defects in RNA processing proteins, such as SMN protein, may lead to the Spinal

malign

Formation of Radicalsn’s disease increased levels of biomarkers of oxidative and nitrosative stress have been identified in the brain. Some of which such as hydroxyalkenals and isoprostanes may even gain diagnostic value for the determination of the degree of neurodegeneration. However, scientific efforts have to be con

Hyperalgesia

Microglia and the Cerebral Defence System or disturbances in the CNS homeostasis. Deregulated or dysfunctional microglia can, on the other hand, be critical or at least instrumental in a harmful way. Excessive and chronic reaction of microglia can then fuel destructive cascades upon trauma or during inflammatory and neurodegenerative proce

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LAP

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