Titlebook: Biomarkers for Huntington‘s Disease; Improving Clinical O Elizabeth A. Thomas,Georgia M. Parkin Book 2023 The Editor(s) (if applicable) and

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書目名稱Biomarkers for Huntington‘s Disease影響因子(影響力)




書目名稱Biomarkers for Huntington‘s Disease影響因子(影響力)學(xué)科排名




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書目名稱Biomarkers for Huntington‘s Disease被引頻次學(xué)科排名




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書目名稱Biomarkers for Huntington‘s Disease讀者反饋學(xué)科排名

北極熊

諂媚于性

,Konflikte in der Wirtschaftsprüfung,with the diagnosis and management of this disorder. This chapter then reviews the different classes of CSF biomarkers that have been studied in HD, including neurotransmitters, amino acids, proteins, metabolites, metals, and microRNAs. CSF levels of mutant huntingtin and neurofilament light chain ar

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,Konflikte in der Wirtschaftsprüfung,apter will review findings from the literature that have explored saliva as a non-invasive biofluid for biomarker research in Huntington’s disease and have identified factors, such as huntingtin, cortisol, uric acid and cytokines, in saliva that track with features of the disease. These studies offe

farewell

EWE

Wolfgang H. C. Junge,Martina Jungedetecting early HD pathophysiology before clinical onset. This chapter will provide an overview of the functional and physiological MRI studies conducted in the premanifest and early-stage HD patients, as well as in preclinical HD models, focusing on discussing the potential of different MRI measure

亞當(dāng)心理陰影

發(fā)微光

Nonflammable

Konflikte um die Aneignung von Landmutant huntingtin within inflammatory cells have been demonstrated. As circulating markers of inflammation has been shown to mirror brain inflammatory changes in HD, this has raised the possibility that these markers could be useful as markers of disease features. This chapter aims to summarize curr

草率男

Beitr?ge zum Diversity ManagementHowever, several case reports of individuals with the HTT mutation and ALS-like syndrome suggest potential interactions and involvement of TDP-43 in the pathophysiology and phenotype of some individuals with HD. Investigations into other PolyQ diseases, such as Spinocerebellar Ataxias, have provided