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Titlebook: Biology of Cellular Transducing Signals; Jack Y. Vanderhoek Book 1990 Plenum Press, New York 1990 Calcium.Colon.DNA.Nucleotide.cyclin.enzy

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發(fā)表于 2025-3-21 17:02:12 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱Biology of Cellular Transducing Signals
影響因子2023Jack Y. Vanderhoek
視頻videohttp://file.papertrans.cn/188/187687/187687.mp4
學(xué)科分類Gwumc Department of Biochemistry and Molecular Biology Annual Spring Symposia
圖書(shū)封面Titlebook: Biology of Cellular Transducing Signals;  Jack Y. Vanderhoek Book 1990 Plenum Press, New York 1990 Calcium.Colon.DNA.Nucleotide.cyclin.enzy
影響因子Intercellular communication in multicellular organisms enables cells to respond to environmental changes. Intercellular signal transduction transmitters include hormones, peptide growth factors, neurotransmitters as well as some lipid-soluble mediators. Once signalling molecules are bound to their cell surface receptors, one or more intracellular signals are generated which alter the Qehavior of the target cell. The IXth International Washington Spring Symposium at the George Washington University in Washington, D.C. was organized to assess the current status of the field of signal transduction processes and regulatory mechanisms. The symposium was held on May 8-‘.2, 1989 and was attended by more than 1000 scientists from 30 countries. Most of the papers presented at the plenary sessions have been collected in this volume. The first section of this book details the action and regulation of receptors such as ,B-pdr.‘nergic receptors and receptors for EGF, insulin, leukotrienes, phosphoinositides and prostaglandins. Section two focuses on the family of guanine nucleotide regulatory proteins (G proteins). These G proteins are known to mediate the coupling of receptor-mediated signals
Pindex Book 1990
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Identification of Residues Essential for the Active Conformation of the ?-Adrenergic Receptor by Sitide binding regulatory proteins (G-proteins). Activation of the G-protein by the agonist-occupied form of the receptor causes the release of GDP from the G-protein, allowing GTP to bind. The GTP-bound form of the G-protein is the active form, which dissociates from the receptor, leading to the activ
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The Role of Covalent and Non-Covalent Mechanisms in Insulin Receptor Actionommonly in insulin and non-insulin dependent diabetes mellitus, but also in obesity, uremia, glucocorticoid and growth hormone excess, and in certain rare syndromes such as lipoatrophic diabetes and leprechaunism (Kahn, 1985; Reaven, 1988). Although insulin action has been intensively investigated a
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EGF Triggers a Similar Signalling Cascade in Different Cell Types Overexpressing the EGF Receptorintracellular second messengers. EGF. is regarded as a classical example of a growth factor which acts on a receptor harboring tyrosine kinase activity (Berridge, 1987 a,b; Carpenter, 1987). Recently, several reports have indicated that in certain cell types, EGF stimulates inositol lipid turnover w
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Second Messenger Receptors in the Phosphoinositide Cycleterm changes in humoral transmission, the PI cycle may regulate long term alterations evoked by growth factors, hormones and in learning and memory, as is exemplified by long term potentiation. The PI cycle influences cellular function through the generation of diacylglycerol (DAG) and inositol tris
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The Family of G Proteinsar effectors, are a family of heterotrimeric proteins composed of α, ? and γ subunits (reviewed by Gilman, 1987; Lochrie and Simon, 1988; Neer and Clapham, 1988). Each of these subunits consists of a super gene family with many members. The α subunits bind guanine nucleotides and all have an intrins
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