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Titlebook: Biology of Brain Dysfunction; Volume 3 Gerald E. Gaull Book 1975 Plenum Press, New York 1975 biology.brain.cortex.growth.information.molecu

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樓主: FLAK
31#
發(fā)表于 2025-3-26 21:43:16 | 只看該作者
32#
發(fā)表于 2025-3-27 02:01:46 | 只看該作者
Kommunikation von Aufsichtsratsvorsitzendeniated with this form of energy have focused the attention of both the scientific community as well as the public on an understanding of the biological effects of these radiations. The beneficial uses of atomic energy such as the development of nuclear power plants and the use of radiations in medica
33#
發(fā)表于 2025-3-27 06:50:53 | 只看該作者
Kommunikation von Aufsichtsratsvorsitzendenl data available in a number of surveys. (.. also Table 1). A more recent study conducted under the auspices of the World Health Organization and reported by Stevenson ... showed the total incidence of major congenital malformations in 411,405 births pooled from different surveys to be 12.7/1000 bir
34#
發(fā)表于 2025-3-27 11:05:49 | 只看該作者
35#
發(fā)表于 2025-3-27 14:24:52 | 只看該作者
36#
發(fā)表于 2025-3-27 20:28:41 | 只看該作者
Theorie und Empirie Lebenslangen Lernensbeen an explosion of information in neurobiology. However, information relating these two areas—i.e., how the enzymatic deficiency results in the brain disease—has not been clear-cut. Indeed, in no instance are we able to relate clearly the inherited enzymatic defect with the neurological deficit.
37#
發(fā)表于 2025-3-28 01:57:36 | 只看該作者
Sequenzen der Kommunikation von Lernen,gressive multifocal leukoencephalopathy (PML). In animals recent additions to the list include mink encephalopathy and Aleutian mink disease. The number of diseases of the central nervous system (CNS) of man that may be caused by an infectious agent and that therefore would probably be placed in the
38#
發(fā)表于 2025-3-28 03:21:58 | 只看該作者
39#
發(fā)表于 2025-3-28 08:00:01 | 只看該作者
40#
發(fā)表于 2025-3-28 10:46:02 | 只看該作者
Pathogenesis of Brain Dysfunction in Inborn Errors of Amino Acid Metabolism,been an explosion of information in neurobiology. However, information relating these two areas—i.e., how the enzymatic deficiency results in the brain disease—has not been clear-cut. Indeed, in no instance are we able to relate clearly the inherited enzymatic defect with the neurological deficit.
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