Titlebook: Asthma Treatment; A Multidisciplinary D. Olivieri,P. J. Barnes,G. C. Folco Book 1992 Springer Science+Business Media New York 1992 Atmen.P

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HALO

勉勵

Injury and Repair of the Airway Epithelium in Asthma,rized by a marked accumulation of acute inflammatory cells including eosinophils and neutrophils (5-11). Both of these cell types possess a considerable armamentarium of proteases, oxidants and toxic molecules. There are, therefore, a number of mechanisms by which airway epithelial cell damage could

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Allergen Inhalation Increases Cough Sensitivity in Subjects with Allergic Asthma, heterogeneity of the populations studied, which included non-allergic and allergic asthmatics. Furthermore, subjects with allergic asthma were sensitized to allergens with different seasonal distribution. Therefore, we hypothesized that allergen exposure increases airway sensitivity to cough stimul

寵愛

https://doi.org/10.1007/978-3-642-57865-6thma, it is not a feature of mild stable disease as evidenced by studies of the numbers of tryptase positive mast cells and by morphometry of cells examined by electron microscopy. Release of cytokines, particularly IL-5, by lymphocytes, favour selective eosinophil adhesion to endothelium and their

Endometrium

不滿分子

,Spannung, Verzerrung, Elastizit?tsgesetz,rized by a marked accumulation of acute inflammatory cells including eosinophils and neutrophils (5-11). Both of these cell types possess a considerable armamentarium of proteases, oxidants and toxic molecules. There are, therefore, a number of mechanisms by which airway epithelial cell damage could

割公牛膨脹

https://doi.org/10.1007/978-3-662-07290-5clinical findings. Indeed, these and other studies have indirectly (by using variables derived from the classical three-compartment lung model, such as the venous admixture ratio or the physiologic dead space) recognised ventilation-perfusion (V./ Q) mismatching as the principal mechanism underlying

surrogate