Titlebook: Arterial Disorders; Definition, Clinical Adel Berbari,Giuseppe Mancia Book 2015 Springer International Publishing Switzerland 2015 Arterial

ACME

Adel Berbari,Giuseppe ManciaDiscusses in detail the pathophysiologic mechanisms of arterial disease.Explains a range of important novel concepts.Covers arterial disease in systemic disorders.Explores the role of macrocirculation

Breach

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kyphoplasty

https://doi.org/10.1007/978-3-319-14556-3Arterial ageing; Calcium phosphorus and bone metabolism; Macro-/microcirculation cross-talk; Renal resi

affinity

978-3-319-35348-7Springer International Publishing Switzerland 2015

resistant

Pennee Narot,Narong Kiettikunwongstimated 17.3 million people died from these conditions which is 48 % of noncommunicable diseases; of these, 6.2 million were a consequence of stroke and 7.3 million due to coronary artery disease [3]. In the UK alone, 160,000 people died of CVD in 2001 [4]. The number of deaths from CVDs is predict

Constant

Pennee Narot,Narong Kiettikunwongial tree to the arterioles and capillaries (microcirculation) to meet the metabolic requirements of surrounding tissues. The walls of arteries are composed of living cells, including smooth muscles cells (SMC), stiff collagen fibers, and compliant elastic fibers. The proximal aorta has large amounts

聯(lián)合

https://doi.org/10.1007/978-981-16-3326-3esponse resulting in sub-endothelial lipoprotein retention and endothelial activation with migration of immune and smooth muscle cells to the inflamed intima. This leads to formation of atheromas with chronic vascular stenosis and tissue ischemia which is often complicated by acute atherothrombotic

愚蠢人

Pennee Narot,Narong Kiettikunwongover 100 years ago. At that time, atherosclerosis was called nodular arteriosclerosis, and arteriosclerosis, as we now know it, was called senile arteriosclerosis. Arteriosclerosis is caused by fatigue and fracture of elastin laminae in the proximal aorta. The proximal thoracic aorta is principally

馬具

Equity in Health Services for Ageinghe renin-angiotensin-aldosterone system, endothelin, catecholamines, substances produced in perivascular fat, and inflammatory and immune mediators such as lymphocytes and macrophages and their products. The vascular phenotype of hypertension varies according to the age of subjects. In younger indiv

Hectic