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Titlebook: Arterial Disorders; Definition, Clinical Adel Berbari,Giuseppe Mancia Book 2015 Springer International Publishing Switzerland 2015 Arterial

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發(fā)表于 2025-3-21 17:24:33 | 只看該作者 |倒序瀏覽 |閱讀模式
期刊全稱Arterial Disorders
期刊簡稱Definition, Clinical
影響因子2023Adel Berbari,Giuseppe Mancia
視頻videohttp://file.papertrans.cn/162/161886/161886.mp4
發(fā)行地址Discusses in detail the pathophysiologic mechanisms of arterial disease.Explains a range of important novel concepts.Covers arterial disease in systemic disorders.Explores the role of macrocirculation
圖書封面Titlebook: Arterial Disorders; Definition, Clinical Adel Berbari,Giuseppe Mancia Book 2015 Springer International Publishing Switzerland 2015 Arterial
影響因子As our knowledge?about arterial disease is greatly expanding, the aim of this book is to explore all aspects of arterial pathology, including classification, clinical manifestations, pathogenesis, and therapeutic options. The discussion of pathophysiologic mechanisms of arterial disease is wide ranging, encompassing hemodynamic, metabolic, humoral, inflammatory, genetic and environmental factors. Particular emphasis is placed on recent concepts, such as: the role of age-associated arterial alterations in the initiation and progression of cardiovascular diseases in older persons, the importance of mineral metabolism-bone vascular interactions, the clinical and prognostic significance of the renal resistive index, retinal circulation, toxemia of pregnancy as an arterial disease, and the role of pulmonary/vascular interaction in pulmonary hypertension and cross-talk of macrocirculation and microcirculation in target organ involvement. Evaluation procedures are carefully explained, and the full range of currently available therapeutic options, including lifestyle modifications and pharmacologic approaches, are described and appraised. ? ?
Pindex Book 2015
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Atherosclerosisesponse resulting in sub-endothelial lipoprotein retention and endothelial activation with migration of immune and smooth muscle cells to the inflamed intima. This leads to formation of atheromas with chronic vascular stenosis and tissue ischemia which is often complicated by acute atherothrombotic
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Vascular Changes in the Microcirculation: Arterial Remodeling and Capillary Rarefactionhe renin-angiotensin-aldosterone system, endothelin, catecholamines, substances produced in perivascular fat, and inflammatory and immune mediators such as lymphocytes and macrophages and their products. The vascular phenotype of hypertension varies according to the age of subjects. In younger indiv
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發(fā)表于 2025-3-22 15:28:39 | 只看該作者
Perinatal Programming of Cardiovascular Diseaseoriginally proposed the hypothesis that several chronic adult diseases can be programmed in early life. These authors examined the mortality records of 5,654 men who were born during 1911–1930 in Hertfordshire (UK). In this report was found that the men with the lowest weights at birth subsequently
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發(fā)表于 2025-3-22 20:21:48 | 只看該作者
Hemodynamic and Mechanical Factors Acting on Arteriesss. Tensile stress corresponds to changes in dimension according to changes in forces applied on the vessel. Shear stress is of a different nature; it corresponds to the friction of viscous fluid (here the blood) on the inner surface of the vessel (here the endothelium). It is to be noted that direc
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發(fā)表于 2025-3-22 21:46:37 | 只看該作者
Role of Metabolic Factors: Lipids, Glucose/Insulin Intoleranced the cardiorenal syndrome (CRS). Obesity, insulin resistance, and diabetes mellitus are associated with a substantially increased prevalence of arterial disease (AD), which is involved in the risk of coronary, cerebral, and peripheral atherosclerosis and the clinical consequences of myocardial infa
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發(fā)表于 2025-3-23 08:25:55 | 只看該作者
Neurohormonal Interactionsogically interact with each other and participate in the regulation of cardiac as well as vascular function. Both endothelial and sympathetic functions seem to be the main important regulatory mechanisms involved in maintaining the homeostatic balance and participating in the cardiovascular response
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