Titlebook: Angiotensin II Receptor Blockade Physiological and Clinical Implications; Naranjan S. Dhalla (Distinguished Professor and Di Book 1998 Spr

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Book 1998ndicated that therapies currently in use for hypertension may have direct application to the treatment of heart failure. The Manitoba Cardiovascular Forum on Angiotensin Receptor Blockade in Winnipeg was convened October 18-20, 1996 to examine the clinical and basic aspects of angiotensin receptor b

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The Brain Renin-Angiotensin System And Salt-Sensitive Hypertensions, peptides, and receptors of the brain RAS and the organization of angiotensinergic pathways involved in cardiovascular regulation. Centrally administered exogenous angiotensin (Ang) II increases sympathetic neuronal activity, decreases the gain of the baroreflex, and induces vasopressin release. A

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At1 Angiotensin Receptor Blockade and Angiotensin Converting Enzyme Inhibition: Effects on Vascular istar-Kyoto control rats (WKY). These differences may be reduced by treatment with angiotensin I-converting enzyme inhibitors (ACEI). It is unclear whether this beneficial effect is the result of inhibition of the generation of angiotensin (Ang) II by ACEI or the result of increased bradykinin accum

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Cellular Physiology of Angiotensin II Receptors in Vascular Smooth Muscle Cells. Regardless of its origin, the response of cardiovascular tissues to Ang II is mediated by specific cell surface receptors. In vascular tissues, two angiotensin receptors have been characterized according to their sensitivity to the specific antagonists losartan and PD123319 [1]. The AT. receptor (

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Angiotensin II Enhanced The Expression Of Inhibitory Guanine Nucleotide Regulatory Protein in Vasculnctions in A-10 vascular smooth muscle cells (VSMCs). Ang II treatment of VSMC enhanced the levels of inhibitory guanine nucleotide regulatory protein (Gi) as well as Gi mRNA in a concentration-dependent manner as determined by immunoblot and Northern blot analysis, respectively. However, the GTPγS-

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