Titlebook: Anesthesiology and the Cardiovascular Patient; Papers presented at T. H. Stanley,P. L. Bailey Book 1996 Kluwer Academic Publishers 1996 an

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https://doi.org/10.1007/978-3-8349-8036-6ay be early markers of coronary heart disease, hypertensive heart disease, and many cardiomyopathies. As many as 40% of patients presenting with cardiac failure may have intact systolic function, implicating diastolic dysfunction as the major cause of failure (1). In addition to ischemia, hypertroph

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endoscopy

https://doi.org/10.1007/978-3-663-13483-1a (1,2). In all groups of patients with silent myocardial ischemia, the risk of cardiovascular events is increased. Silent ischemia may result from an imbalance between increased oxygen demand and limited supply (demand ischemia) or from an acute reduction in oxygen supply (supply ischemia). Why it

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J. D. Auffermann,Ph. Kreuzer,K. Schwantagt ventricle/interventricular septum. The right ventricular chamber anatomically and functionally consists of an inflow portion, the “body,” and an outflow tract. Although the left and right ventricles appear as almost separate entities, they are bound together by common, spiraling muscle bundles (1)

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Dynamische Computertomographie,disease, with one out of every two deaths attributable to cardiovascular disease, and with more than one-third of the healthcare resources appropriated to the diagnosis and treatment of cardiovascular disease. The impact of cardiovascular disease on the surgical patients is substantial, as well. Reg

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morale

Lagerbestand und Nachschubstrategien,e of solid clot. This view has been fostered by medical school teaching that has focused upon the coagulation cascades. The effects of platelet numbers and platelet function may be less well understood and monitored. The complexity of coagulation and its many nuances can be daunting.

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